The genetic basis of aortic aneurysm

Cold Spring Harbor Perspectives in Medicine

Volumn 4, Issue 9, 2014, Pages

  Lindsay, Mark E ^a   Dietz, Harry C ^b,c  

^a MASSACHUSETTS GENERAL HOSPITAL   (United States)

^b JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c HOWARD HUGHES MEDICAL INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CELL SURFACE RECEPTOR; COLLAGEN TYPE 3; ELASTIN; FIBRILLIN; FIBRILLIN 1; FIBULIN 4; FIBULIN 5; FILAMIN A; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 3; MONOCYTE CHEMOTACTIC PROTEIN 1; NEUTRALIZING ANTIBODY; PHOSPHATIDYLINOSITOL 3 KINASE; SMAD2 PROTEIN; THROMBOSPONDIN 1; TRANSFORMING GROWTH FACTOR BETA; TRANSFORMING GROWTH FACTOR BETA2; UNCLASSIFIED DRUG; CYTOKINE; DNA;

AORTA ANEURYSM; ARACHNODACTYLY; ARTICLE; BASIC RESEARCH; CYTOSKELETON; DISEASE COURSE; DISEASE SEVERITY; ELASTIC FIBER; EMBRYONIC STEM CELL; ENZYME PHOSPHORYLATION; GENE DELETION; GENE IDENTIFICATION; GENE MUTATION; GENETIC ASSOCIATION; GENETICS; HAPLOTYPE; HUMAN; HYPERTROPHIC CARDIOMYOPATHY; INTRACELLULAR SIGNALING; LOSS OF FUNCTION MUTATION; LUNG EMPHYSEMA; MARFAN SYNDROME; NONHUMAN; PLURIPOTENT STEM CELL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; PSEUDOXANTHOMA ELASTICUM; SIGNAL TRANSDUCTION; SMOOTH MUSCLE CONTRACTION; SPONTANEOUS PNEUMOTHORAX; UPREGULATION; ANIMAL; BIOSYNTHESIS; GENE EXPRESSION REGULATION; GENETIC PREDISPOSITION;

ANIMALIA;

ANIMALS; AORTIC ANEURYSM; CYTOKINES; DISEASE PROGRESSION; DNA; GENE EXPRESSION REGULATION; GENETIC PREDISPOSITION TO DISEASE; HUMANS; SIGNAL TRANSDUCTION;

EID: 84923886344     PISSN: None     EISSN: 21571422     Source Type: Journal    
DOI: 10.1101/cshperspect.a015909     Document Type: Article

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