Cigarette smoke extract affects mitochondrial function in alveolar epithelial cells

American Journal of Physiology - Lung Cellular and Molecular Physiology

Volumn 307, Issue 11, 2014, Pages L895-L907

  Ballweg, Korbinian ^a   Mutze, Kathrin ^a   Königshoff, Melanie ^a   Eickelberg, Oliver ^a   Meiners, Silke ^a  

^a LUDWIG MAXIMILIANS UNIVERSITY   (Germany)

Author keywords

Chronic obstructive pulmonary disease; Emphysema; Proteostasis; Stress induced mitochondrial hyperfusion

Indexed keywords

CIGARETTE SMOKE; MITOCHONDRIAL PROTEIN; MITOFUSIN 2; GUANOSINE TRIPHOSPHATASE; MFN2 PROTEIN, MOUSE; SMOKE;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CELL SURVIVAL; CELLULAR STRESS RESPONSE; CHRONIC OBSTRUCTIVE LUNG DISEASE; CONTROLLED STUDY; EXTRACT; GENE EXPRESSION; LUNG ALVEOLUS EPITHELIUM; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; MITOPHAGY; MOUSE; NONHUMAN; OXYGEN CONSUMPTION; PATHOGENESIS; PROTEIN EXPRESSION; PROTEIN HOMEOSTASIS; UNFOLDED PROTEIN RESPONSE; ADVERSE EFFECTS; ANIMAL; BIOSYNTHESIS; C57BL MOUSE; CELL LINE; CHEMICALLY INDUCED; DRUG EFFECTS; INFLAMMATION; LUNG; LUNG ALVEOLUS; OXIDATIVE STRESS; PATHOLOGY; PULMONARY DISEASE, CHRONIC OBSTRUCTIVE; RESPIRATORY TRACT MUCOSA; SMOKE; SMOKING; TOBACCO; ULTRASTRUCTURE;

ANIMALS; CELL LINE; GTP PHOSPHOHYDROLASES; INFLAMMATION; LUNG; MEMBRANE POTENTIAL, MITOCHONDRIAL; MICE; MICE, INBRED C57BL; MITOCHONDRIA; MITOCHONDRIAL DEGRADATION; OXIDATIVE STRESS; OXYGEN CONSUMPTION; PULMONARY ALVEOLI; PULMONARY DISEASE, CHRONIC OBSTRUCTIVE; RESPIRATORY MUCOSA; SMOKE; SMOKING; TOBACCO; UNFOLDED PROTEIN RESPONSE;

EID: 84913553177     PISSN: 10400605     EISSN: 15221504     Source Type: Journal    
DOI: 10.1152/ajplung.00180.2014     Document Type: Article

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