Neutrophils counteract autophagy-mediated anti-inflammatory mechanisms in alveolar macrophage: Role in posthemorrhagic shock acute lung inflammation

Journal of Immunology

Volumn 193, Issue 9, 2014, Pages 4623-4633

  Wen, Zongmei ^a,b,c   Fan, Liyan ^d   Li, Yuehua ^c   Zou, Zui ^a   Scott, Melanie J ^c   Xiao, Guozhi ^e   Li, Song ^d   Billiar, Timothy R ^c,d   Wilson, Mark A ^c,f   Shi, Xueyin ^a   Fan, Jie ^c,d,f  

^a SECOND MILITARY MEDICAL UNIVERSITY   (China)

^b TONGJI UNIVERSITY SCHOOL OF MEDICINE   (China)

^c UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

^d UNIVERSITY OF PITTSBURGH   (United States)

^e RUSH UNIVERSITY MEDICAL CENTER   (United States)

^f VETERANS AFFAIRS MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE RECRUITMENT DOMAIN PROTEIN 15; HIGH MOBILITY GROUP B1 PROTEIN; I KAPPA B KINASE GAMMA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INFLAMMASOME; MURAMYL DIPEPTIDE; NUCLEOTIDE BINDING OLIGOMERIZATION DOMAIN PROTEIN; NUCLEOTIDE BINDING OLIGOMERIZATION DOMAIN PROTEIN 3; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; TOLL LIKE RECEPTOR 4; UNCLASSIFIED DRUG; CYTOKINE;

ACUTE LUNG INJURY; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; AUTOPHAGY; CELL INTERACTION; CELL MIGRATION; CONTROLLED STUDY; ENZYME PHOSPHORYLATION; FEEDBACK SYSTEM; HEMORRHAGIC SHOCK; INFLAMMATION; LUNG ALVEOLUS MACROPHAGE; MALE; MOUSE; NEUTROPHIL; NONHUMAN; PNEUMONIA; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; UPREGULATION; ANIMAL; COMPLICATION; DISEASE MODEL; GENE EXPRESSION; GENETICS; IMMUNOLOGY; KNOCKOUT MOUSE; MACROPHAGE ACTIVATION; METABOLISM; PATHOLOGY;

ACUTE LUNG INJURY; ANIMALS; AUTOPHAGY; CYTOKINES; DISEASE MODELS, ANIMAL; GENE EXPRESSION; HMGB1 PROTEIN; INFLAMMASOMES; INFLAMMATION; MACROPHAGE ACTIVATION; MACROPHAGES, ALVEOLAR; MALE; MICE; MICE, KNOCKOUT; NADPH OXIDASE; NEUTROPHILS; NF-KAPPA B; NOD2 SIGNALING ADAPTOR PROTEIN; SHOCK, HEMORRHAGIC; SIGNAL TRANSDUCTION; TOLL-LIKE RECEPTOR 4;

EID: 84908124134     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1400899     Document Type: Article

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