Complement-mediated glomerular injury is reduced by inhibition of protein-tyrosine phosphatase 1B

American Journal of Physiology - Renal Physiology

Volumn 307, Issue 5, 2014, Pages

  Nezvitsky, Lisa ^a   Tremblay, Michel L ^a   Takano, Tomoko ^a   Papillon, Joan ^a   Cybulsky, Andrey V ^a  

^a MCGILL UNIVERSITY HEALTH CENTRE   (Canada)

Author keywords

Cytotoxicity; Endoplasmic reticulum; Podocytes

Indexed keywords

PROTEIN TYROSINE PHOSPHATASE 1B; COMPLEMENT; PTPN1 PROTEIN, MOUSE; PTPN1 PROTEIN, RAT;

ANIMAL CELL; ARTICLE; COMPLEMENT DEPENDENT CYTOTOXICITY; CONTROLLED STUDY; ENDOPLASMIC RETICULUM ASSOCIATED DEGRADATION; ENZYME ACTIVITY; ENZYME INHIBITION; EPITHELIUM CELL; FIBROBLAST; GENETIC TRANSFECTION; GLOMERULOPATHY; GLOMERULUS; MEMBRANOUS GLOMERULONEPHRITIS; MOUSE; NONHUMAN; PODOCYTE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; PROTEINURIA; UNFOLDED PROTEIN RESPONSE; ANIMAL; ANTAGONISTS AND INHIBITORS; BAGG ALBINO MOUSE; CELL CULTURE; DEFICIENCY; DISEASE MODEL; DRUG EFFECTS; GLOMERULONEPHRITIS; KNOCKOUT MOUSE; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; RAT;

ANIMALS; CELLS, CULTURED; COMPLEMENT SYSTEM PROTEINS; DISEASE MODELS, ANIMAL; ENDOPLASMIC RETICULUM-ASSOCIATED DEGRADATION; EPITHELIAL CELLS; FIBROBLASTS; GLOMERULONEPHRITIS; KIDNEY GLOMERULUS; MICE; MICE, INBRED BALB C; MICE, KNOCKOUT; PODOCYTES; PROTEIN TYROSINE PHOSPHATASE, NON-RECEPTOR TYPE 1; RATS; UNFOLDED PROTEIN RESPONSE;

EID: 84906897946     PISSN: 1931857X     EISSN: 15221466     Source Type: Journal    
DOI: 10.1152/ajprenal.00191.2014     Document Type: Article

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