AMPK-dependent inhibitory phosphorylation of ACC is not essential for maintaining myocardial fatty acid oxidation

Circulation Research

Volumn 115, Issue 5, 2014, Pages 518-524

  Zordoky, Beshay N M ^a   Nagendran, Jeevan ^a   Pulinilkunnil, Thomas ^a   Kienesberger, Petra C ^a   Masson, Grant ^a   Waller, Terri J ^a   Kemp, Bruce E ^b   Steinberg, Gregory R ^c   Dyck, Jason R B ^a  

^a UNIVERSITY OF ALBERTA   (Canada)

^b UNIVERSITY OF MELBOURNE   (Australia)

^c MCMASTER UNIVERSITY   (Canada)

Author keywords

acetyl CoA carboxylase; AMP activated protein kinase

Indexed keywords

ACETYL COENZYME A CARBOXYLASE; CYCLIC AMP DEPENDENT PROTEIN KINASE; FATTY ACID TRANSPORT PROTEIN 1; FATTY ACID TRANSPORTER; HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE KINASE; MALONYL COENZYME A; MITOGEN ACTIVATED PROTEIN KINASE P38; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR ALPHA; PROTEIN KINASE B; TRIACYLGLYCEROL LIPASE; UNCLASSIFIED DRUG; UNCOUPLING PROTEIN 3; ACACB PROTEIN, MOUSE; FATTY ACID; SERINE;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENERGY METABOLISM; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME INACTIVATION; FATTY ACID OXIDATION; FEMALE; GENOTYPE; GLUCOSE METABOLISM; HEART EJECTION FRACTION; HEART FUNCTION; HEART MUSCLE; HEART MUSCLE ISCHEMIA; MALE; METABOLIC STRESS; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; ANIMAL; ENZYMOLOGY; GENE TARGETING; GENETICS; HEART CONTRACTION; HEART LEFT VENTRICLE FUNCTION; METABOLISM; MUTANT MOUSE STRAIN; MUTATION; OXIDATION REDUCTION REACTION; PHOSPHORYLATION; TIME; TRANSGENIC MOUSE;

ACETYL-COA CARBOXYLASE; AMP-ACTIVATED PROTEIN KINASES; ANIMALS; ENERGY METABOLISM; FATTY ACIDS; FEMALE; GENE KNOCK-IN TECHNIQUES; MALE; MALONYL COENZYME A; MICE, MUTANT STRAINS; MICE, TRANSGENIC; MUTATION; MYOCARDIAL CONTRACTION; MYOCARDIUM; OXIDATION-REDUCTION; PHOSPHORYLATION; SERINE; TIME FACTORS; VENTRICULAR FUNCTION, LEFT;

EID: 84906227277     PISSN: 00097330     EISSN: 15244571     Source Type: Journal    
DOI: 10.1161/CIRCRESAHA.115.304538     Document Type: Article

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