NOD2 is dispensable for ATG16L1 deficiency-mediated resistance to urinary tract infection

Autophagy

Volumn 10, Issue 2, 2014, Pages 331-338

  Wang, Caihong ^a   Yuan, Xuejun ^a   Emily, Ma ^a   Mendonsa, Graziella R ^a   Plantinga, Theo S ^b   Kiemeney, Lambertus A ^b   Vermeulen, Sita H ^b   Mysorekar, Indira U ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b RADBOUD UNIVERSITY MEDICAL CENTER   (Netherlands)

Author keywords

Bladder; Crohn disease; G908R; Kidney; L1007finsC; NLR; Pyelonephritis; R702W; Uropathogenic E. coli

Indexed keywords

ATG16L1 PROTEIN; CASPASE RECRUITMENT DOMAIN PROTEIN 15; LYSOSOME ASSOCIATED MEMBRANE PROTEIN 1; PROTEIN; UNCLASSIFIED DRUG;

ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; AUTOPHAGY; BACTERIAL CLEARANCE; BACTERIAL COLONIZATION; BACTERIAL LOAD; BLADDER MUCOSA; CONTROLLED STUDY; DISEASE ASSOCIATION; DISEASE COURSE; FEMALE; GENETIC POLYMORPHISM; HOST RESISTANCE; IMMUNOCOMPETENT CELL; INCIDENCE; KIDNEY ABSCESS; MOUSE; NEUTROPHIL COUNT; NONHUMAN; PATHOGENESIS; PROTEIN DEPLETION; PROTEIN FUNCTION; RENAL PROTECTION; URINARY TRACT; URINARY TRACT INFECTION; UROPATHOGENIC ESCHERICHIA COLI;

BLADDER; CROHN DISEASE; G908R; KIDNEY; L1007FINSC; NLR; PYELONEPHRITIS; R702W; UROPATHOGENIC E. COLI;

ANIMALS; AUTOPHAGY; CARRIER PROTEINS; CROHN DISEASE; DISEASE MODELS, ANIMAL; FEMALE; GENETIC PREDISPOSITION TO DISEASE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NOD2 SIGNALING ADAPTOR PROTEIN; URINARY TRACT INFECTIONS;

EID: 84894486754     PISSN: 15548627     EISSN: 15548635     Source Type: Journal    
DOI: 10.4161/auto.27196     Document Type: Article

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