Transmaternal bisphenol a exposure accelerates diabetes type 1 development in NOD mice

Toxicological Sciences

Volumn 137, Issue 2, 2014, Pages 311-323

  Bodin, Johanna ^a   Bølling, Anette Kocbach ^a   Becher, Rune ^a   Kuper, Frieke ^b   Løvik, Martinus ^a,c   Nygaard, Unni Cecilie ^a  

^a NORWEGIAN INSTITUTE OF PUBLIC HEALTH   (Norway)

^b TNO   (Netherlands)

^c NORWEGIAN UNIVERSITY OF SCIENCE AND TECHNOLOGY   (Norway)

Author keywords

Bisphenol A; Developmental toxicity; Diabetes mellitus type 1; Immunotoxicity; Insulitis; NOD mice

Indexed keywords

4,4' ISOPROPYLIDENEDIPHENOL; CASPASE 3; CHAPERONIN 60; GLUCAGON; GLUCOSE; GLUTAMATE DECARBOXYLASE 65; INSULIN; INTERLEUKIN 10; INTERLEUKIN 17; INTERLEUKIN 2; TESTOSTERONE; THYROXINE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL INFILTRATION; CONTROLLED STUDY; CYTOKINE RELEASE; DISEASE COURSE; ENVIRONMENTAL EXPOSURE; FEMALE; GLUCOSE BLOOD LEVEL; INSULIN DEPENDENT DIABETES MELLITUS; INSULITIS; LYMPH NODE; LYMPH NODE CELL; MACROPHAGE; MALE; MOUSE; NATURAL KILLER T CELL; NONHUMAN; NONOBESE DIABETIC MOUSE; PANCREAS ISLET ALPHA CELL; PROGENY; REGULATORY T LYMPHOCYTE; SPLEEN CELL; TESTOSTERONE BLOOD LEVEL; THYROXINE BLOOD LEVEL;

BISPHENOL A; DEVELOPMENTAL TOXICITY; DIABETES MELLITUS TYPE 1; IMMUNOTOXICITY; INSULITIS; NOD MICE;

ANIMALS; APOPTOSIS; BENZHYDRYL COMPOUNDS; BLOOD GLUCOSE; CELL PROLIFERATION; CYTOKINES; DIABETES MELLITUS, TYPE 1; ENDOCRINE DISRUPTORS; FEMALE; ISLETS OF LANGERHANS; MACROPHAGES; MATERNAL EXPOSURE; MICE; MICE, INBRED NOD; PHENOLS; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; SPLEEN; T-LYMPHOCYTES, REGULATORY;

EID: 84893374706     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kft242     Document Type: Article

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