Long-term bisphenol A exposure accelerates insulitis development in diabetes-prone NOD mice

Immunopharmacology and Immunotoxicology

Volumn 35, Issue 3, 2013, Pages 349-358

  Bodin, Johanna ^a   Bolling, Anette Kocbach ^a   Samuelsen, Mari ^a   Becher, Rune ^a   Lovik, Martinus ^a,b   Nygaard, Unni Cecilie ^a  

^a NORWEGIAN INSTITUTE OF PUBLIC HEALTH   (Norway)

^b NORWEGIAN UNIVERSITY OF SCIENCE AND TECHNOLOGY   (Norway)

Author keywords

Bisphenol A; Endocrine disruptor; Pancreatic beta cells; Type 1 diabetes mellitus

Indexed keywords

4,4' ISOPROPYLIDENEDIPHENOL; DRINKING WATER; GAMMA INTERFERON; GAMMA INTERFERON INDUCIBLE PROTEIN 10; GLUCOSE; GLUTAMATE DECARBOXYLASE 65 ANTIBODY; INSULIN ANTIBODY; INTERLEUKIN 10; INTERLEUKIN 1BETA; MONOCYTE CHEMOTACTIC PROTEIN 1; TRANSFORMING GROWTH FACTOR BETA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL FUNCTION; CONTROLLED STUDY; CYTOKINE RELEASE; CYTOTOXIC T LYMPHOCYTE; DIABETOGENESIS; DISEASE COURSE; DISEASE SEVERITY; FEMALE; GLUCOSE BLOOD LEVEL; HYPERGLYCEMIA; IMMUNOHISTOCHEMISTRY; INSULIN BLOOD LEVEL; INSULIN DEPENDENT DIABETES MELLITUS; INSULITIS; LONG TERM EXPOSURE; MACROPHAGE; MALE; MOUSE; NONHUMAN; NONOBESE DIABETIC MOUSE; PANCREAS ISLET; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; REGULATORY T LYMPHOCYTE; TISSUE SECTION; URINALYSIS;

ANIMALS; APOPTOSIS; AUTOANTIBODIES; BENZHYDRYL COMPOUNDS; BLOOD GLUCOSE; CYTOKINES; DIABETES MELLITUS, EXPERIMENTAL; DIABETES MELLITUS, TYPE 1; DOSE-RESPONSE RELATIONSHIP, DRUG; ENDOCRINE DISRUPTORS; FEMALE; GENE-ENVIRONMENT INTERACTION; GENETIC PREDISPOSITION TO DISEASE; INSULIN; INSULIN-SECRETING CELLS; MICE; MICE, INBRED NOD; PANCREAS; PHENOLS;

EID: 84877869198     PISSN: 08923973     EISSN: 15322513     Source Type: Journal    
DOI: 10.3109/08923973.2013.772195     Document Type: Article

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