Infarct size reduction by cyclosporine A at reperfusion involves inhibition of the mitochondrial permeability transition pore but does not improve mitochondrial respiration

Archives of Medical Science

Volumn 9, Issue 6, 2013, Pages 968-975

  Gedik, Nilguen ^a   Heusch, Gerd ^a   Skyschally, Andreas ^a  

^a UNIVERSITY HOSPITAL ESSEN   (Germany)

Author keywords

Cyclosporine A; Infarct size; Ischemic postconditioning; Mitochondrion; Myocardial ischemia reperfusion

Indexed keywords

CALCINEURIN; CALCIUM; CYCLOSPORIN A; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; PHOSPHATASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DRUG INFUSION; HEART INFARCTION; HEART INFARCTION SIZE; HEART MUSCLE CELL; HEART MUSCLE REPERFUSION; HEART PROTECTION; ISCHEMIC POSTCONDITIONING; MINIPIG; MITOCHONDRIAL RESPIRATION; NONHUMAN;

EID: 84892401777     PISSN: 17341922     EISSN: 18969151     Source Type: Journal    
DOI: 10.5114/aoms.2013.38175     Document Type: Article

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