Pretreatment of SH-SY5Y cells with dicaffeoylquinic acids attenuates the reduced expression of nicotinic receptors, elevated level of oxidative stress and enhanced apoptosis caused by β-amyloid peptide

Journal of Pharmacy and Pharmacology

Volumn 65, Issue 12, 2013, Pages 1736-1744

  Deng, Jie ^a   Qi, Xiao Lan ^a   Guan, Zhi Zhong ^a   Yan, Xiu Ming ^a   Huang, Yong ^a   Wang, Yong Lin ^a  

^a GUIZHOU MEDICAL UNIVERSITY   (China)

Author keywords

amyloid peptide; apoptosis; dicaffeoylquinic acids; lipid peroxidation; nicotinic acetylcholine receptor

Indexed keywords

3,4 DICAFFEOYLQUINIC ACID; AMYLOID BETA PROTEIN[25-35]; BUNGAROTOXIN RECEPTOR; ERIGERON BREVISCAPUS EXTRACT; ISOCHLOROGENIC ACID; NEUROPROTECTIVE AGENT; NICOTINIC RECEPTOR; NICOTINIC RECEPTOR ALPHA3; RECEPTOR SUBUNIT; SUPEROXIDE DISMUTASE; UNCLASSIFIED DRUG; XANTHINE OXIDASE;

APOPTOSIS; ARTICLE; CHINESE MEDICINE; CONTROLLED STUDY; ERIGERON; ERIGERON BREVISCAPUS; FLOW CYTOMETRY; HUMAN; HUMAN CELL; IN VITRO STUDY; LIPID PEROXIDATION; NEUROBLASTOMA CELL; NEUROPROTECTION; NEUROTOXICITY; OXIDATIVE STRESS; PROTEIN EXPRESSION; WESTERN BLOTTING;

APOPTOSIS; DICAFFEOYLQUINIC ACIDS; LIPID PEROXIDATION; NICOTINIC ACETYLCHOLINE RECEPTOR; Β-AMYLOID PEPTIDE;

AMYLOID BETA-PEPTIDES; APOPTOSIS; CELL LINE, TUMOR; HUMANS; LIPID PEROXIDATION; NEUROBLASTOMA; NEUROPROTECTIVE AGENTS; OXIDATIVE STRESS; QUINIC ACID; REACTIVE OXYGEN SPECIES; RECEPTORS, NICOTINIC; SUPEROXIDE DISMUTASE;

EID: 84887947434     PISSN: 00223573     EISSN: 20427158     Source Type: Journal    
DOI: 10.1111/jphp.12096     Document Type: Article

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