Molecular contributions to neurovascular unit dysfunctions after brain injuries: Lessons for target-specific drug development

Future Neurology

Volumn 8, Issue 6, 2013, Pages 677-689

  Jullienne, Amandine ^a   Badaut, Jérôme ^a  

^a LOMA LINDA UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

aquaporin 4; blood brain barrier; cerebral blood flow; edema; expanded neurovascular unit; ischemic stroke; matrix metalloproteinases; nitric oxide; tissue plasminogen activator; traumatic brain injury

Indexed keywords

ACETAZOLAMIDE; AQUAPORIN 1; AQUAPORIN 4; AQUAPORIN 9; BUMETANIDE; CLAUDIN 5; ENDOTHELIAL NITRIC OXIDE SYNTHASE; ERYTHROPOIETIN; GELATINASE A; GELATINASE B; INDUCIBLE NITRIC OXIDE SYNTHASE; METHAZOLAMIDE; MULTIDRUG RESISTANCE PROTEIN 2; NEURONAL NITRIC OXIDE SYNTHASE; NITRIC OXIDE; NITROPRUSSIDE SODIUM; STROMELYSIN; TISSUE PLASMINOGEN ACTIVATOR; VALPROIC ACID;

ALZHEIMER DISEASE; BLOOD BRAIN BARRIER; BRAIN BLOOD FLOW; BRAIN EDEMA; BRAIN HEMORRHAGE; BRAIN ISCHEMIA; BRAIN PERFUSION; CEREBROVASCULAR ACCIDENT; DRUG MECHANISM; EXPANDED NEUROVASCULAR UNIT; HUMAN; NEUROMODULATION; NEUROPATHOLOGY; NEUROPHYSIOLOGY; NEUROPROTECTION; NONHUMAN; OUTCOME ASSESSMENT; OXIDATIVE STRESS; PATHOPHYSIOLOGY; PHENOTYPIC VARIATION; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; REVIEW; RISK FACTOR; TRAUMATIC BRAIN INJURY;

EID: 84887235583     PISSN: 14796708     EISSN: 17486971     Source Type: Journal    
DOI: 10.2217/fnl.13.55     Document Type: Review

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