P62 expression and autophagy in αB-crystallin R120G mutant knock-in mouse model of hereditary cataract

Experimental Eye Research

Volumn 115, Issue , 2013, Pages 263-273

  Wignes, Jonathan A ^a   Goldman, Joshua W ^a   Weihl, Conrad C ^a   Bartley, Matthew G ^a   Andley, Usha P ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

B crystallin; Autophagy; Cataracts; P62 protein; Protein degradation

Indexed keywords

ALPHA CRYSTALLIN; PROTEIN P62;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; AUTOPHAGOSOME; AUTOPHAGY; CATARACT; CATARACTOGENESIS; CELL NUCLEUS MEMBRANE; CONTROLLED STUDY; CYTOPLASM; EMBRYONIC STEM CELL; EPITHELIUM CELL; GENETIC DISORDER; HETEROZYGOTE; HOMOLOGOUS RECOMBINATION; IMMUNOHISTOCHEMISTRY; LENS; MOLECULAR WEIGHT; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN AGGREGATION; PROTEIN DEGRADATION; PROTEIN EXPRESSION; TRANSMISSION ELECTRON MICROSCOPY; WILD TYPE;

ΑB-CRYSTALLIN; AUTOPHAGY; CATARACTS; P62 PROTEIN; PROTEIN DEGRADATION;

ALPHA-CRYSTALLIN B CHAIN; ANIMALS; AUTOPHAGY; CATARACT; DISEASE MODELS, ANIMAL; EPITHELIAL CELLS; FLUORESCENT ANTIBODY TECHNIQUE, INDIRECT; GENE EXPRESSION REGULATION; GENOTYPE; IMMUNOBLOTTING; LENS, CRYSTALLINE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICROSCOPY, ELECTRON, TRANSMISSION; PHAGOSOMES; POINT MUTATION; POLYMERASE CHAIN REACTION; TRANSCRIPTION FACTORS;

EID: 84883615399     PISSN: 00144835     EISSN: 10960007     Source Type: Journal    
DOI: 10.1016/j.exer.2013.06.026     Document Type: Article

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