The lack of a functional p21WAF1/CIP1 gene ameliorates progression to chronic renal failure

Proceedings of the National Academy of Sciences of the United States of America

Volumn 96, Issue 19, 1999, Pages 10830-10835

  Megyesi, Judit ^a   Price, Peter M ^a   Tamayo, Esther ^b   Safirstein, Robert L ^a  

^a UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

^b University of Texas Medical Branch School of Medicine   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN P21;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; CELL CYCLE; CHRONIC KIDNEY FAILURE; DISEASE COURSE; GENE EXPRESSION; KIDNEY FAILURE; KIDNEY MASS; MOUSE; NONHUMAN; PRIORITY JOURNAL;

ANIMALS; BLOOD PRESSURE; BODY WEIGHT; CELL CYCLE; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; DISEASE MODELS, ANIMAL; GLOMERULOSCLEROSIS, FOCAL SEGMENTAL; HYPERTROPHY; IN SITU HYBRIDIZATION; INSULIN; KIDNEY; KIDNEY FAILURE; MALE; MICE; MUTAGENESIS; TIME FACTORS;

ANIMALIA; RODENTIA;

EID: 0032875388     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.96.19.10830     Document Type: Article

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