Bile acid signal-induced phosphorylation of small heterodimer partner by protein kinase Cζ is critical for epigenomic regulation of liver metabolic genes

Journal of Biological Chemistry

Volumn 288, Issue 32, 2013, Pages 23252-23263

  Seok, Sunmi ^a   Kanamaluru, Deepthi ^a   Xiao, Zhen ^b   Ryerson, Daniel ^a   Choi, Sung E ^a   Suino Powell, Kelly ^c   Xu, H Eric ^c   Veenstra, Timothy D ^b   Kemper, Jongsook Kim ^a  

^a UNIVERSITY OF ILLINOIS AT URBANA CHAMPAIGN   (United States)

^b SAIC FREDERICK INC   (United States)

^c VAN ANDEL RESEARCH INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BILE ACID; HETERO DIMERS; HISTONE MODIFICATION; METABOLIC GENES; PROTEIN KINASE C; THERAPEUTIC TARGETS;

BIOMOLECULES; ENZYMES; GENES; METABOLISM; PHOSPHORYLATION; PROTEINS;

BODY FLUIDS;

BILE ACID; HETERODIMER; PROTEIN KINASE C ZETA; SMALL HETERODIMER PARTNER; TRIACYLGLYCEROL; UNCLASSIFIED DRUG;

ANIMAL TISSUE; ARTICLE; CELL NUCLEUS; CELLULAR DISTRIBUTION; CHROMATIN ASSEMBLY AND DISASSEMBLY; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME PHOSPHORYLATION; EPIGENETICS; GENE MUTATION; GENE TARGETING; HISTONE MODIFICATION; LIPID LIVER LEVEL; LIVER METABOLISM; MALE; METABOLIC REGULATION; METABOLIC SYNDROME X; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN METHYLATION; SIGNAL TRANSDUCTION;

BILE ACID; CDCA; CYP7A1; EPIGENOMICS; FGF19; FIBROBLAST GROWTH FACTOR (FGF); HISTONE MODIFICATION; LIVER METABOLISM; POST-TRANSLATIONAL MODIFICATION;

ANIMALS; BILE ACIDS AND SALTS; EPIGENESIS, GENETIC; FIBROBLAST GROWTH FACTORS; HEP G2 CELLS; HUMANS; LIVER; MALE; METHYLATION; MICE; MICE, INBRED BALB C; MUTATION; PHOSPHORYLATION; PROTEIN KINASE C-EPSILON; RECEPTORS, CYTOPLASMIC AND NUCLEAR; SIGNAL TRANSDUCTION;

EID: 84881457500     PISSN: 00219258     EISSN: 1083351X     Source Type: Journal    
DOI: 10.1074/jbc.M113.452037     Document Type: Article

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