Impact of trichostatin A and sodium valproate treatment on post-stroke neurogenesis and behavioral outcomes in immature mice

Frontiers in Cellular Neuroscience

Volumn , Issue JUL, 2013, Pages

  George, S ^a   Kadam, S D ^a,b   Irving, N D ^a   Markowitz, G J ^a   Raja, S ^a   Kwan, A ^a   Tu, Y ^a   Chen, H ^a   Rohde, C ^c   Smith, D R ^d   Comi, Anne M ^a,b  

^a KENNEDY KRIEGER INSTITUTE   (United States)

^b JOHNS HOPKINS UNIVERSITY   (United States)

^c JOHNS HOPKINS BLOOMBERG SCHOOL OF PUBLIC HEALTH   (United States)

^d JOHNS HOPKINS UNIVERSITY   (United States)

Author keywords

Anticonvulsants; Behavioral outcomes; Hippocampal neurogenesis; Histone deacetylase inhibitors; Neonatal stroke; Trichostatin A; Valproate

Indexed keywords

HISTONE DEACETYLASE; TRICHOSTATIN A; VALPROIC ACID;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BEHAVIORAL SCIENCE; BRAIN ATROPHY; BRAIN NERVE CELL; CEREBROVASCULAR ACCIDENT; COGNITIVE DEFECT; CONTROLLED STUDY; DENTATE GYRUS; DRUG BLOOD LEVEL; DRUG EFFECT; ENZYME INHIBITION; MORTALITY; MOUSE; NERVOUS SYSTEM DEVELOPMENT; NONHUMAN; SEIZURE; WEIGHT GAIN;

EID: 84880157131     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2013.00123     Document Type: Article

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