Mechanism of capsaicin receptor TRPV1-mediated toxicity in pain-sensing neurons focusing on the effects of Na+/Ca2+ fluxes and the Ca2+-binding protein calretinin

Biochimica et Biophysica Acta - Molecular Cell Research

Volumn 1833, Issue 7, 2013, Pages 1680-1691

  Pecze, László ^a   Blum, Walter ^a   Schwaller, Beat ^a  

^a UNIVERSITY OF FRIBOURG   (Switzerland)

Author keywords

Calcium binding proteins; Calretinin; Membrane bleb formation; Sensory neurons; TRPV1

Indexed keywords

CALCIUM ION; CALRETININ; SODIUM ION; VANILLOID RECEPTOR 1;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CELL DEATH; CELL LOSS; CELL MEMBRANE; CELL SWELLING; CONTROLLED STUDY; EMBRYO; NERVE CELL; NOCICEPTION; NONHUMAN; PRIORITY JOURNAL; RAT; SODIUM CALCIUM EXCHANGE; SPINAL GANGLION; WATER TRANSPORT;

ANIMALS; BLOTTING, WESTERN; CALCIUM; CALCIUM-BINDING PROTEIN, VITAMIN D-DEPENDENT; CAPSAICIN; CELL MEMBRANE; CELL SIZE; GANGLIA, SPINAL; IMMUNOENZYME TECHNIQUES; MICE; NEURONS; NIH 3T3 CELLS; PAIN; RATS; SENSORY SYSTEM AGENTS; SODIUM; TRPV CATION CHANNELS;

RATTUS;

EID: 84878242479     PISSN: 01674889     EISSN: 18792596     Source Type: Journal    
DOI: 10.1016/j.bbamcr.2012.08.018     Document Type: Article

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