AMPA receptor-mediated excitotoxicity in human NT2-N neurons results from loss of intracellular Ca2+ homeostasis following marked elevation of intracellular Na+

Journal of Neurochemistry

Volumn 71, Issue 1, 1998, Pages 112-124

  Itoh, Takayuki ^a   Itoh, Aki ^a   Horiuchi, Kazumi ^a   Pleasure, David ^a  

^a CHILDREN S HOSPITAL OF PHILADELPHIA   (United States)

Author keywords

Calcium; Glutamate excitotoxicity; Human neuron; Sodium; Sodium calcium exchange; Amino 3 hydroxy 5 methylisoxazole 4 propionic acid receptor desensitization

Indexed keywords

ALPHA AMINO 3 HYDROXY 5 METHYL 4 ISOXAZOLEPROPIONIC ACID; AMPA RECEPTOR; CYCLOTHIAZIDE; DIZOCILPINE; GLUTAMIC ACID;

ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CELL DEATH; CELL LOSS; CELL MEMBRANE PERMEABILITY; CELL VIABILITY; EXTRACELLULAR CALCIUM; GLIA CELL; HUMAN; HUMAN CELL; MITOCHONDRIAL MEMBRANE; NEUROTRANSMITTER RELEASE; PRIORITY JOURNAL; RECEPTOR DOWN REGULATION; SODIUM CALCIUM EXCHANGE; SODIUM CELL LEVEL;

EID: 0031814379     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.1998.71010112.x     Document Type: Article

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