Accumulation of the parkin substrate, FAF1, plays a key role in the dopaminergic neurodegeneration

Human Molecular Genetics

Volumn 22, Issue 8, 2013, Pages 1558-1573

  Sul, Jee Won ^a   Park, Min Young ^a   Shin, Juhee ^b   Kim, Young Ran ^a   Yoo, Sung Eun ^a   Kong, Young Yun ^b   Kwon, Ki Sun ^c   Lee, Young Ho ^a   Kim, Eunhee ^a  

^a Chungnam National University   (South Korea)

^b Seoul National University   (South Korea)

^c Korea Research Institute of Bioscience and Biotechnology (KRIBB)   (South Korea)

Author keywords

[No Author keywords available]

Indexed keywords

1,2,3,6 TETRAHYDRO 1 METHYL 4 PHENYLPYRIDINE; FAS ASSOCIATED DEATH DOMAIN PROTEIN; FAS ASSOCIATED FACTOR 1; PARKIN; UBIQUITIN PROTEIN LIGASE E3; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL DEATH; CONTROLLED STUDY; DOPAMINERGIC NERVE CELL; DOPAMINERGIC TRANSMISSION; IN VITRO STUDY; LOCOMOTION; LOSS OF FUNCTION MUTATION; MALE; MOUSE; MUTAGENESIS; NERVE DEGENERATION; NONHUMAN; PARKINSON DISEASE; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN PROTEIN INTERACTION; SUBSTANTIA NIGRA PARS COMPACTA; UBIQUITINATION;

ANIMALS; CARRIER PROTEINS; DOPAMINERGIC NEURONS; HUMANS; MICE; MOTOR ACTIVITY; MUTATION; NERVE DEGENERATION; PARKINSON DISEASE; PARKINSONIAN DISORDERS; PROTEOLYSIS; UBIQUITIN-PROTEIN LIGASES; UBIQUITINATION;

MUS;

EID: 84875791139     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/ddt006     Document Type: Article

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