The late Na+ current - Origin and pathophysiological relevance

Cardiovascular Drugs and Therapy

Volumn 27, Issue 1, 2013, Pages 61-68

  Zaza, Antonio ^a   Rocchetti, Marcella ^a  

^a UNIVERSITY OF MILANO BICOCCA   (Italy)

Author keywords

Arrhythmias; Ca2+ mishandling; INaL; Metabolic dysfunction; Repolarization reserve

Indexed keywords

ANKYRIN; CALCIUM CALMODULIN DEPENDENT PROTEIN KINASE; CALCIUM ION; CAVEOLIN 3; POTASSIUM CHANNEL HERG; POTASSIUM ION; REACTIVE OXYGEN METABOLITE; RYANODINE RECEPTOR; SODIUM CALCIUM EXCHANGE PROTEIN; SODIUM CHANNEL; SODIUM CHANNEL NAV1.5; SODIUM CHANNEL NAV1.8; SODIUM ION; SODIUM PROTON EXCHANGE PROTEIN;

ARRHYTHMOGENESIS; CELL MEMBRANE DEPOLARIZATION; CHANNEL GATING; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ENZYME ACTIVATION; GENE MUTATION; HEART ARRHYTHMIA; HEART MUSCLE CELL; HEART MUSCLE FIBER MEMBRANE POTENTIAL; HEART REPOLARIZATION; HUMAN; LONG QT SYNDROME; MOUSE; NONHUMAN; PATHOPHYSIOLOGY; POTASSIUM CURRENT; PRIORITY JOURNAL; RABBIT; RAT; REVIEW; SODIUM CHANNELOPATHY; SODIUM CURRENT;

ACTION POTENTIALS; ANIMALS; ARRHYTHMIAS, CARDIAC; CALCIUM; HEART VENTRICLES; HUMANS; MYOCYTES, CARDIAC; SODIUM CHANNEL BLOCKERS; SODIUM CHANNELS;

EID: 84873058846     PISSN: 09203206     EISSN: 15737241     Source Type: Journal    
DOI: 10.1007/s10557-012-6430-0     Document Type: Review

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