Two mechanisms involved in trigeminal CGRP release: Implications for migraine treatment

Headache

Volumn 53, Issue 1, 2013, Pages 67-80

  Durham, Paul L ^a   Masterson, Caleb G ^a  

^a MISSOURI STATE UNIVERSITY   (United States)

Author keywords

acid sensitive ion channel; calcitonin gene related peptide; onabotulinumtoxinA; proton; trigeminal; triptan

Indexed keywords

BOTULINUM TOXIN A; CALCITONIN GENE RELATED PEPTIDE; CAPSAZEPINE; RIZATRIPTAN; SODIUM ION; TRIPTAN DERIVATIVE;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; EXTRACELLULAR CALCIUM; FEMALE; IMMUNOHISTOCHEMISTRY; IMMUNOREACTIVITY; MIGRAINE; NOCICEPTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; SENSITIZATION; SENSORY NERVE CELL; SODIUM CELL LEVEL; TRANSFORMED MIGRAINE; TRIGEMINUS GANGLION;

ANALGESICS; ANIMALS; BLOTTING, WESTERN; BOTULINUM TOXINS, TYPE A; CALCITONIN GENE-RELATED PEPTIDE; CELLS, CULTURED; IMMUNOHISTOCHEMISTRY; MIGRAINE DISORDERS; NEURONS; RADIOIMMUNOASSAY; RATS; RATS, SPRAGUE-DAWLEY; TRIAZOLES; TRIGEMINAL GANGLION; TRYPTAMINES;

EID: 84872118507     PISSN: 00178748     EISSN: 15264610     Source Type: Journal    
DOI: 10.1111/j.1526-4610.2012.02262.x     Document Type: Article

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