Dissecting the different biological effects of oncogenic Ras isoforms in cancer cell lines: Could stimulation of oxidative stress be the one more weapon of H-Ras? Regulation of oxidative stress and Ras biological effects

Medical Hypotheses

Volumn 79, Issue 6, 2012, Pages 731-734

  Bellavia, Maurizio ^a,b   Gioviale, Maria Concetta ^b   Damiano, Giuseppe ^b   Palumbo, Vincenzo Davide ^b   Spinelli, Gabriele ^b   Buscemi, Giuseppe ^a,b   Lo Monte, Attilio Ignazio ^a,b  

^a UNIVERSITY OF PALERMO   (Italy)

^b UNIVERSITY OF PALERMO   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

ISOPROTEIN; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHATIDYLINOSITOL 3 KINASE; RAS PROTEIN; REACTIVE OXYGEN METABOLITE;

ANGIOGENESIS; ARTICLE; CANCER CELL CULTURE; CANCER GROWTH; CANCER INVASION; CELL ADHESION; CELL ORGANELLE; ENZYME ACTIVATION; HUMAN; METASTASIS; ONCOGENE H RAS; ONCOGENE N RAS; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PHENOTYPE; POINT MUTATION; PROTEIN LOCALIZATION; REGULATORY MECHANISM;

CELL LINE, TUMOR; GENES, RAS; HUMANS; MODELS, THEORETICAL; ONCOGENES; OXIDATIVE STRESS;

EID: 84869083397     PISSN: 03069877     EISSN: 15322777     Source Type: Journal    
DOI: 10.1016/j.mehy.2012.08.012     Document Type: Article

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