Gα q and Gα 11 contribute to the maintenance of cellular electrophysiology and Ca 2+ handling in ventricular cardiomyocytes

Cardiovascular Research

Volumn 95, Issue 1, 2012, Pages 48-58

  Pahlavan, Sara ^a   Oberhofer, Martin ^a   Sauer, Benjamin ^a   Ruppenthal, Sandra ^a   Tian, Qinghai ^a   Scholz, Anke ^a   Kaestner, Lars ^a   Lipp, Peter ^a  

^a SAARLAND UNIVERSITY   (Germany)

Author keywords

Ca 2+ homoeostasis; Cellular electrophysiology; Cre recombinase; G q G 11; Tamoxifen

Indexed keywords

CALCIUM; CRE RECOMBINASE; GUANINE NUCLEOTIDE BINDING PROTEIN; PROTEIN GALPHA 11; PROTEIN GALPHA Q; TAMOXIFEN; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CELL FUNCTION; CONTROLLED STUDY; HEART MUSCLE CELL; HEART MUSCLE FIBER MEMBRANE POTENTIAL; HEART VENTRICLE MUSCLE FIBER; MEMBRANE CURRENT; MOUSE; NONHUMAN; PATCH CLAMP; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PURKINJE FIBER;

ACTION POTENTIALS; ANIMALS; CALCIUM; GTP-BINDING PROTEIN ALPHA SUBUNITS, GQ-G11; HEART VENTRICLES; INTEGRASES; MICE; MICE, TRANSGENIC; MYOCYTES, CARDIAC; TAMOXIFEN;

EID: 84862842299     PISSN: 00086363     EISSN: 17553245     Source Type: Journal    
DOI: 10.1093/cvr/cvs162     Document Type: Article

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