Postconditioning modulates ischemia-damaged mitochondria during reperfusion

Journal of Cardiovascular Pharmacology

Volumn 59, Issue 1, 2012, Pages 101-108

  Chen, Qun ^a   Paillard, Melanie ^b   Gomez, Ludovic ^b   Li, Heng ^a   Hu, Ying ^a   Lesnefsky, Edward J ^a,c,d  

^a VIRGINIA COMMONWEALTH UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b UNIVERSITÉ LYON 1   (France)

^c VIRGINIA COMMONWEALTH UNIVERSITY   (United States)

^d VETERANS AFFAIRS MEDICAL CENTER   (United States)

Author keywords

electron transport chain; mitochondria; mitochondrial permeability transition pore; reperfusion

Indexed keywords

AMOBARBITAL;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL DAMAGE; CELL ISOLATION; CELL PROTECTION; CONTROLLED STUDY; ELECTRON TRANSPORT; HEART MUSCLE INJURY; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; ISCHEMIC POSTCONDITIONING; ISOLATED HEART; MALE; MITOCHONDRION; NONHUMAN; OXIDATIVE PHOSPHORYLATION; PRIORITY JOURNAL; RAT; REPERFUSION INJURY; SARCOLEMMA; SIGNAL TRANSDUCTION;

AMOBARBITAL; ANIMALS; ELECTRON TRANSPORT; ISCHEMIC POSTCONDITIONING; MALE; MEMBRANE POTENTIAL, MITOCHONDRIAL; MITOCHONDRIA, HEART; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; OXIDATIVE PHOSPHORYLATION; RATS; RATS, INBRED F344;

EID: 84855359223     PISSN: 01602446     EISSN: 15334023     Source Type: Journal    
DOI: 10.1097/FJC.0b013e31823827cc     Document Type: Article

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