Increased glyoxalase I levels inhibit accumulation of oxidative stress and an advanced glycation end product in mouse mesangial cells cultured in high glucose

Experimental Cell Research

Volumn 318, Issue 2, 2012, Pages 152-159

  Kim, Ki Mo ^a   Kim, Young Sook ^a   Jung, Dong Ho ^a   Lee, Jun ^a   Kim, Jin Sook ^a  

^a Korea Institute of Oriental Medicine (KIOM)   (South Korea)

Author keywords

Advanced glycation end product; Glyoxalase I; High glucose; Methylgloxal; Oxidative stress

Indexed keywords

8 HYDROXYDEOXYGUANOSINE; ADENOSINE DIPHOSPHATE RIBOSE; ADVANCED GLYCATION END PRODUCT; CASPASE 3; DNA DIRECTED DNA POLYMERASE BETA; DNA NUCLEOTIDYLEXOTRANSFERASE; GLUCOSE; LACTOYLGLUTATHIONE LYASE; METHYLGLYOXAL; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); SUCCINATE DEHYDROGENASE (UBIQUINONE); UBIQUINOL CYTOCHROME C REDUCTASE;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL CULTURE; CONTROLLED STUDY; DIABETES MELLITUS; DNA CLEAVAGE; DNA DAMAGE; ENZYME BLOOD LEVEL; JUXTAGLOMERULAR CELL; LIPID PEROXIDATION; MESANGIUM CELL; MOUSE; NICK END LABELING; NITROSATIVE STRESS; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION;

EID: 82855177408     PISSN: 00144827     EISSN: 10902422     Source Type: Journal    
DOI: 10.1016/j.yexcr.2011.10.013     Document Type: Article

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