-
3
-
-
22544478489
-
Increased risk of common infections in patients with type 1 and type 2 diabetes mellitus
-
Muller LM, et al. (2005) Increased risk of common infections in patients with type 1 and type 2 diabetes mellitus. Clin. Infect. Dis. 41:281-8.
-
(2005)
Clin. Infect. Dis.
, vol.41
, pp. 281-288
-
-
Muller, L.M.1
-
4
-
-
0041308233
-
Quantifying the risk of infectious diseases for people with diabetes
-
Shah BR, Hux JE. (2003) Quantifying the risk of infectious diseases for people with diabetes. Diabetes Care. 26:510-3.
-
(2003)
Diabetes Care
, vol.26
, pp. 510-513
-
-
Shah, B.R.1
Hux, J.E.2
-
5
-
-
0031035272
-
Impaired leucocyte functions in diabetic patients
-
Delamaire M, et al. (1997) Impaired leucocyte functions in diabetic patients. Diabet. Med. 14:29-34.
-
(1997)
Diabet. Med.
, vol.14
, pp. 29-34
-
-
Delamaire, M.1
-
6
-
-
0028880606
-
Neutrophil bactericidal function in diabetes mellitus: Evidence for association with blood glucose control
-
Gallacher SJ, et al. (1995) Neutrophil bactericidal function in diabetes mellitus: evidence for association with blood glucose control. Diabet. Med.12:916-20.
-
(1995)
Diabet. Med.
, vol.12
, pp. 916-920
-
-
Gallacher, S.J.1
-
7
-
-
0037184495
-
Molecular understanding of hyperglycemia's adverse effects for diabetic complications
-
Sheetz MJ, King GL. (2002) Molecular understanding of hyperglycemia's adverse effects for diabetic complications. JAMA. 288:2579-88.
-
(2002)
JAMA
, vol.288
, pp. 2579-2588
-
-
Sheetz, M.J.1
King, G.L.2
-
8
-
-
54249117325
-
Hyperglycaemic conditions decrease cultured keratinocyte mobility: Implications for impaired wound healing in patients with diabetes
-
Lan CC, Liu IH, Fang AH, Wen CH, Wu CS. (2008) Hyperglycaemic conditions decrease cultured keratinocyte mobility: implications for impaired wound healing in patients with diabetes. Br. J. Dermatol. 159:1103-15.
-
(2008)
Br. J. Dermatol.
, vol.159
, pp. 1103-1115
-
-
Lan, C.C.1
Liu, I.H.2
Fang, A.H.3
Wen, C.H.4
Wu, C.S.5
-
9
-
-
66149144785
-
Hyperglycaemic conditions hamper keratinocyte locomotion via sequential inhibition of distinct pathways: New insights on poor wound closure in patients with diabetes
-
Lan CC, Wu CS, Kuo HY, Huang SM, Chen GS. (2009) Hyperglycaemic conditions hamper keratinocyte locomotion via sequential inhibition of distinct pathways: new insights on poor wound closure in patients with diabetes. Br. J. Dermatol.160:1206-14.
-
(2009)
Br. J. Dermatol.
, vol.160
, pp. 1206-1214
-
-
Lan, C.C.1
Wu, C.S.2
Kuo, H.Y.3
Huang, S.M.4
Chen, G.S.5
-
13
-
-
0034946112
-
Expression of the peptide antibiotics human beta defensin-1 and human beta defensin-2 in normal human skin
-
Ali RS, et al. (2001) Expression of the peptide antibiotics human beta defensin-1 and human beta defensin-2 in normal human skin. J. Invest. Dermatol.117:106-11.
-
(2001)
J. Invest. Dermatol.
, vol.117
, pp. 106-111
-
-
Ali, R.S.1
-
14
-
-
0034703072
-
CD14-dependent lipopolysaccharideinduced beta-defensin-2 expression in human tracheobronchial epithelium
-
Becker MN, Diamond G, Verghese MW, Randell SH. (2000) CD14-dependent lipopolysaccharideinduced beta-defensin-2 expression in human tracheobronchial epithelium. J. Biol. Chem.275:29731-6.
-
(2000)
J. Biol. Chem.
, vol.275
, pp. 29731-29736
-
-
Becker, M.N.1
Diamond, G.2
Verghese, M.W.3
Randell, S.H.4
-
15
-
-
0031581544
-
Expression of natural peptide antibiotics in human skin
-
Fulton C, Anderson GM, Zasloff M, Bull R, Quinn AG. (1997) Expression of natural peptide antibiotics in human skin. Lancet. 350:1750-1.
-
(1997)
Lancet
, vol.350
, pp. 1750-1751
-
-
Fulton, C.1
Anderson, G.M.2
Zasloff, M.3
Bull, R.4
Quinn, A.G.5
-
16
-
-
3042717148
-
Up-regulation of human beta-defensin 2 by interleukin-1beta in A549 cells: Involvement of PI3K, PKC, p38 MAPK, JNK, and NF-kappaB
-
Jang BC, et al. (2004) Up-regulation of human beta-defensin 2 by interleukin-1beta in A549 cells: involvement of PI3K, PKC, p38 MAPK, JNK, and NF-kappaB. Biochem. Biophys. Res. Commun.320:1026-33.
-
(2004)
Biochem. Biophys. Res. Commun.
, vol.320
, pp. 1026-1033
-
-
Jang, B.C.1
-
17
-
-
0035941493
-
Discovery of new human beta-defensins using a genomics-based approach
-
Jia HP, et al. (2001) Discovery of new human beta-defensins using a genomics-based approach. Gene. 263:211-8.
-
(2001)
Gene
, vol.263
, pp. 211-218
-
-
Jia, H.P.1
-
18
-
-
0000215491
-
Production of betadefensins by human airway epithelia
-
Singh PK, et al. (1998) Production of betadefensins by human airway epithelia. Proc. Natl. Acad. Sci. U. S. A. 95:14961-6.
-
(1998)
Proc. Natl. Acad. Sci. U. S. A.
, vol.95
, pp. 14961-14966
-
-
Singh, P.K.1
-
19
-
-
17044367481
-
Differential regulation of beta-defensin expression in human skin by microbial stimuli
-
Sorensen OE, et al. (2005) Differential regulation of beta-defensin expression in human skin by microbial stimuli. J. Immunol. 174:4870-9.
-
(2005)
J. Immunol.
, vol.174
, pp. 4870-4879
-
-
Sorensen, O.E.1
-
20
-
-
0029927322
-
Diabetic foot infections
-
Bacteriology and activity of 10 oral antimicrobial agents against bacteria isolated from consecutive cases
-
Goldstein EJ, Citron DM, Nesbit CA. (1996) Diabetic foot infections. Bacteriology and activity of 10 oral antimicrobial agents against bacteria isolated from consecutive cases. Diabetes Care. 19:638-41.
-
(1996)
Diabetes Care
, vol.19
, pp. 638-641
-
-
Goldstein, E.J.1
Citron, D.M.2
Nesbit, C.A.3
-
21
-
-
34848918479
-
The constitutive capacity of human keratinocytes to kill Staphylococcus aureus is dependent on beta-defensin 3
-
Kisich KO, et al. (2007) The constitutive capacity of human keratinocytes to kill Staphylococcus aureus is dependent on beta-defensin 3. J. Invest. Dermatol. 127:2368-80.
-
(2007)
J. Invest. Dermatol.
, vol.127
, pp. 2368-2380
-
-
Kisich, K.O.1
-
22
-
-
24344477170
-
FK506 promotes melanocyte and melanoblast growth and creates a favorable milieu for cell migration via keratinocytes: Possible mechanisms of how tacrolimus ointment induces repigmentation in patients with vitiligo
-
Lan CC, et al. (2005) FK506 promotes melanocyte and melanoblast growth and creates a favorable milieu for cell migration via keratinocytes: Possible mechanisms of how tacrolimus ointment induces repigmentation in patients with vitiligo. Br. J. Dermatol. 153:498-505.
-
(2005)
Br. J. Dermatol.
, vol.153
, pp. 498-505
-
-
Lan, C.C.1
-
23
-
-
0032031820
-
Evaluation of a disk diffusion method for determining susceptibility of Mycobacterium avium complex to clarithromycin
-
Jarboe E, et al. (1998) Evaluation of a disk diffusion method for determining susceptibility of Mycobacterium avium complex to clarithromycin. Diagn. Microbiol. Infect. Dis. 30:197-203.
-
(1998)
Diagn. Microbiol. Infect. Dis.
, vol.30
, pp. 197-203
-
-
Jarboe, E.1
-
24
-
-
33745849146
-
Injury-induced innate immune response in human skin mediated by transactivation of the epidermal growth factor receptor
-
Sorensen OE, et al. (2006) Injury-induced innate immune response in human skin mediated by transactivation of the epidermal growth factor receptor. J. Clin. Invest. 116:1878-85.
-
(2006)
J. Clin. Invest.
, vol.116
, pp. 1878-1885
-
-
Sorensen, O.E.1
-
26
-
-
0036890119
-
Expression of functional Toll-like receptor 2 on human epidermal keratinocytes
-
Kawai K, Shimura H, Minagawa M, Ito A, Tomiyama K, Ito M. (2002) Expression of functional Toll-like receptor 2 on human epidermal keratinocytes. J. Dermatol. Sci. 30:185-94.
-
(2002)
J. Dermatol. Sci.
, vol.30
, pp. 185-194
-
-
Kawai, K.1
Shimura, H.2
Minagawa, M.3
Ito, A.4
Tomiyama, K.5
Ito, M.6
-
27
-
-
0038182550
-
Lipoteichoic acid (LTA) of Streptococcus pneumoniae and Staphylococcus aureus activates immune cells via Toll-like receptor (TLR)-2, lipopolysaccharide-binding protein (LBP), and CD14, whereas TLR-4 and MD-2 are not involved
-
Schroder NW, et al. (2003) Lipoteichoic acid (LTA) of Streptococcus pneumoniae and Staphylococcus aureus activates immune cells via Toll-like receptor (TLR)-2, lipopolysaccharide-binding protein (LBP), and CD14, whereas TLR-4 and MD-2 are not involved. J. Biol. Chem. 278:15587-94.
-
(2003)
J. Biol. Chem.
, vol.278
, pp. 15587-15594
-
-
Schroder, N.W.1
-
28
-
-
0037406799
-
Defensin expression by the cornea: Multiple signalling pathways mediate IL-1beta stimulation of hBD-2 expression by human corneal epithelial cells
-
McDermott AM, et al. (2003) Defensin expression by the cornea: multiple signalling pathways mediate IL-1beta stimulation of hBD-2 expression by human corneal epithelial cells. Invest. Ophthalmol. Vis. Sci. 44:1859-65.
-
(2003)
Invest. Ophthalmol. Vis. Sci.
, vol.44
, pp. 1859-1865
-
-
McDermott, A.M.1
-
29
-
-
0035839525
-
Salmonella enteritidis FliC (flagella filament protein) induces human beta-defensin-2 mRNA production by Caco-2 cells
-
Ogushi K, et al. (2001) Salmonella enteritidis FliC (flagella filament protein) induces human beta-defensin-2 mRNA production by Caco-2 cells. J. Biol. Chem. 276:30521-6.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 30521-30526
-
-
Ogushi, K.1
-
30
-
-
0035132062
-
Helicobacter pylori-mediated transcriptional regulation of the human betadefensin 2 gene requires NF-kappaB
-
Wada A, et al. (2001) Helicobacter pylori-mediated transcriptional regulation of the human betadefensin 2 gene requires NF-kappaB. Cell. Microbiol.3:115-23.
-
(2001)
Cell. Microbiol.
, vol.3
, pp. 115-123
-
-
Wada, A.1
-
31
-
-
0029808748
-
Transcription factor AP-1 regulation by mitogen-activated protein kinase signal transduction pathways
-
Whitmarsh AJ, Davis RJ. (1996) Transcription factor AP-1 regulation by mitogen-activated protein kinase signal transduction pathways. J. Mol. Med.74:589-607.
-
(1996)
J. Mol. Med.
, vol.74
, pp. 589-607
-
-
Whitmarsh, A.J.1
Davis, R.J.2
-
32
-
-
0028453652
-
Lilly Lecture 1993. Glycation and diabetic complications
-
Brownlee M. (1994) Lilly Lecture 1993. Glycation and diabetic complications. Diabetes. 43:836-41.
-
(1994)
Diabetes
, vol.43
, pp. 836-841
-
-
Brownlee, M.1
-
33
-
-
33847082865
-
Advanced glycation end products induce apoptosis in fibroblasts through activation of ROS, MAP kinases, and the FOXO1 transcription factor
-
Alikhani M, et al. (2007) Advanced glycation end products induce apoptosis in fibroblasts through activation of ROS, MAP kinases, and the FOXO1 transcription factor. Am. J. Physiol. Cell. Physiol.292:C850-6.
-
(2007)
Am. J. Physiol. Cell. Physiol.
, vol.292
-
-
Alikhani, M.1
-
34
-
-
0037741359
-
Aminosalicylic acid reduces the antiproliferative effect of hyperglycaemia, advanced glycation endproducts and glycated basic fibroblast growth factor in cultured bovine aortic endothelial cells: Comparison with aminoguanidine
-
Duraisamy Y, et al. (2003) Aminosalicylic acid reduces the antiproliferative effect of hyperglycaemia, advanced glycation endproducts and glycated basic fibroblast growth factor in cultured bovine aortic endothelial cells: comparison with aminoguanidine. Mol. Cell. Biochem. 246:143-53.
-
(2003)
Mol. Cell. Biochem.
, vol.246
, pp. 143-153
-
-
Duraisamy, Y.1
-
35
-
-
0031748727
-
Aminoguanidine inhibits reactive oxygen species formation, lipid peroxidation, and oxidant- induced apoptosis
-
Giardino I, Fard AK, Hatchell DL, Brownlee M. (1998) Aminoguanidine inhibits reactive oxygen species formation, lipid peroxidation, and oxidant- induced apoptosis. Diabetes. 47:1114-20.
-
(1998)
Diabetes
, vol.47
, pp. 1114-1120
-
-
Giardino, I.1
Fard, A.K.2
Hatchell, D.L.3
Brownlee, M.4
-
36
-
-
0032708329
-
Aminoguanidine prevents impaired healing and deficient angiogenesis in diabetic rats
-
Teixeira AS, Caliari MV, Rocha OA, Machado RD, Andrade SP. (1999) Aminoguanidine prevents impaired healing and deficient angiogenesis in diabetic rats. Inflammation. 23:569-581
-
(1999)
Inflammation
, vol.23
, pp. 569-581
-
-
Teixeira, A.S.1
Caliari, M.V.2
Rocha, O.A.3
Machado, R.D.4
Andrade, S.P.5
-
37
-
-
28844447845
-
Collagen ultrastructure and TGF-beta1 expression preserved with aminoguanidine during wound healing in diabetic rats
-
Yavuz D, et al. (2005) Collagen ultrastructure and TGF-beta1 expression preserved with aminoguanidine during wound healing in diabetic rats. Endocr. Res. 31:229-43.
-
(2005)
Endocr. Res.
, vol.31
, pp. 229-243
-
-
Yavuz, D.1
-
38
-
-
77949273822
-
p38 MAPK-regulated EGFR internalization takes place in keratinocyte monolayer during stress conditions
-
Lambert S, Frankart A, Poumay Y. (2010) p38 MAPK-regulated EGFR internalization takes place in keratinocyte monolayer during stress conditions. Arch. Dermatol. Res. 302:229-33.
-
(2010)
Arch. Dermatol. Res.
, vol.302
, pp. 229-233
-
-
Lambert, S.1
Frankart, A.2
Poumay, Y.3
-
39
-
-
0030859290
-
Activation of the receptor for advanced glycation end products triggers a p21(ras)-dependent mitogen-activated protein kinase pathway regulated by oxidant stress
-
Lander HM, et al. (1997) Activation of the receptor for advanced glycation end products triggers a p21(ras)-dependent mitogen-activated protein kinase pathway regulated by oxidant stress. J. Biol. Chem. 272:17810-4.
-
(1997)
J. Biol. Chem.
, vol.272
, pp. 17810-17814
-
-
Lander, H.M.1
-
40
-
-
0034987093
-
Requirement for p38 and p44/p42 mitogen-activated protein kinases in RAGE-mediated nuclear factor-kappaB transcriptional activation and cytokine secretion
-
Yeh CH, et al. (2001) Requirement for p38 and p44/p42 mitogen-activated protein kinases in RAGE-mediated nuclear factor-kappaB transcriptional activation and cytokine secretion. Diabetes.50:1495-504.
-
(2001)
Diabetes
, vol.50
, pp. 1495-1504
-
-
Yeh, C.H.1
-
41
-
-
65349178179
-
Human beta-defensin-3 promotes wound healing in infected diabetic wounds
-
Hirsch T, et al. (2009) Human beta-defensin-3 promotes wound healing in infected diabetic wounds. J. Gene. Med. 11:220-8.
-
(2009)
J. Gene. Med.
, vol.11
, pp. 220-228
-
-
Hirsch, T.1
|