Hyperglycaemic conditions hamper keratinocyte locomotion via sequential inhibition of distinct pathways: new insights on poor wound closure in patients with diabetes

British Journal of Dermatology

Volumn 160, Issue 6, 2009, Pages 1206-1214

  Lan, C C E ^a   Wu, C S ^b   Kuo, H Y ^a   Huang, S M ^a   Chen, G S ^a  

^a KAOHSIUNG MEDICAL UNIVERSITY HOSPITAL   (Taiwan)

^b KAOHSIUNG MEDICAL UNIVERSITY   (Taiwan)

Author keywords

Cell migration; Glucose; Keratinocyte; Matrix metalloproteinase 1; Signal transducer and activator of transcription 1

Indexed keywords

COLLAGEN TYPE 1; GLUCOSE; INTERSTITIAL COLLAGENASE; MESSENGER RNA; STAT1 PROTEIN; VERY LATE ACTIVATION ANTIGEN 2;

ARTICLE; CELL LEVEL; CELL MIGRATION; CELL MOTILITY; CONTROLLED STUDY; DIABETES MELLITUS; DOWN REGULATION; DRUG MECHANISM; FLOW CYTOMETRY; HUMAN; HUMAN CELL; HYPERGLYCEMIA; IN VITRO STUDY; KERATINOCYTE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; TREATMENT DURATION; WESTERN BLOTTING; WOUND CLOSURE; WOUND HEALING;

CELL MOVEMENT; CELLS, CULTURED; DIABETES MELLITUS; GLUCOSE; HUMANS; KERATINOCYTES; MATRIX METALLOPROTEINASE 1; STATISTICS AS TOPIC; TISSUE INHIBITOR OF METALLOPROTEINASE-1; WOUND HEALING;

EID: 66149144785     PISSN: 00070963     EISSN: 13652133     Source Type: Journal    
DOI: 10.1111/j.1365-2133.2009.09089.x     Document Type: Article

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