-
1
-
-
0032201606
-
Myocardial dysfunction in sepsis
-
Piper R.D. Myocardial dysfunction in sepsis. Clin Exp Pharmacol Physiol 25 (1998) 951
-
(1998)
Clin Exp Pharmacol Physiol
, vol.25
, pp. 951
-
-
Piper, R.D.1
-
2
-
-
32844463046
-
Sepsis-induced myocardial dysfunction and myocardial protection from ischemia/reperfusion injury
-
McDonough K.H., and Virag J.I. Sepsis-induced myocardial dysfunction and myocardial protection from ischemia/reperfusion injury. Front Biosci 11 (2006) 23
-
(2006)
Front Biosci
, vol.11
, pp. 23
-
-
McDonough, K.H.1
Virag, J.I.2
-
3
-
-
34249326963
-
Mechanisms of sepsis-induced cardiac dysfunction
-
Rudiger A., and Mervyn S. Mechanisms of sepsis-induced cardiac dysfunction. Crit Care Med 35 (2007) 1599
-
(2007)
Crit Care Med
, vol.35
, pp. 1599
-
-
Rudiger, A.1
Mervyn, S.2
-
4
-
-
34447106947
-
Distinct cardiodynamic and molecular characteristics during early and late stages of sepsis-induced myocardial dysfunction
-
Chopra M., and Sharma A.C. Distinct cardiodynamic and molecular characteristics during early and late stages of sepsis-induced myocardial dysfunction. Life Sci 81 (2007) 306
-
(2007)
Life Sci
, vol.81
, pp. 306
-
-
Chopra, M.1
Sharma, A.C.2
-
5
-
-
17244372648
-
Big endothelin-1 (1-21) fragment during early sepsis modulates tau, p38-MAPK phosphorylation and nitric oxide synthase activation
-
Brahmbhatt S., Gupta A., and Sharma A.C. Big endothelin-1 (1-21) fragment during early sepsis modulates tau, p38-MAPK phosphorylation and nitric oxide synthase activation. Mol Cell Biochem 271 (2005) 225
-
(2005)
Mol Cell Biochem
, vol.271
, pp. 225
-
-
Brahmbhatt, S.1
Gupta, A.2
Sharma, A.C.3
-
6
-
-
0032546638
-
Doppler tissue imaging quantitates regional wall motion during myocardial ischemia and reperfusion
-
Derumeaux G., Ovize M., Loufoua J., et al. Doppler tissue imaging quantitates regional wall motion during myocardial ischemia and reperfusion. Circulation 97 (1989) 1970
-
(1989)
Circulation
, vol.97
, pp. 1970
-
-
Derumeaux, G.1
Ovize, M.2
Loufoua, J.3
-
8
-
-
22944443625
-
Molecular mechanisms involved in the pathogenesis of septic shock
-
Lopez-Bojorquez L.N., Dehesa A.Z., and Reyes-Teran G. Molecular mechanisms involved in the pathogenesis of septic shock. Arch Med Res 35 (2004) 465
-
(2004)
Arch Med Res
, vol.35
, pp. 465
-
-
Lopez-Bojorquez, L.N.1
Dehesa, A.Z.2
Reyes-Teran, G.3
-
9
-
-
0025060845
-
Right ventricular dysfunction and dilatation, similar to left ventricular changes, characterize the cardiac depression of septic shock in humans
-
Parker M.M., McCarthy K.E., Ognibene F.P., et al. Right ventricular dysfunction and dilatation, similar to left ventricular changes, characterize the cardiac depression of septic shock in humans. Chest 97 (1999) 126
-
(1999)
Chest
, vol.97
, pp. 126
-
-
Parker, M.M.1
McCarthy, K.E.2
Ognibene, F.P.3
-
10
-
-
0036892344
-
Clinical review: Myocardial depression in sepsis and septic shock
-
Court O., Kumar A., Parrillo J.E., et al. Clinical review: Myocardial depression in sepsis and septic shock. Crit Care 6 (2002) 500
-
(2002)
Crit Care
, vol.6
, pp. 500
-
-
Court, O.1
Kumar, A.2
Parrillo, J.E.3
-
12
-
-
0031918203
-
Tumor necrosis factor in the heart
-
Meldrum D.R. Tumor necrosis factor in the heart. Am J Physiol 274 (1998) R577
-
(1998)
Am J Physiol
, vol.274
-
-
Meldrum, D.R.1
-
13
-
-
34249326963
-
Mechanisms of sepsis-induced cardiac dysfunction
-
Rudiger A., and Singer M. Mechanisms of sepsis-induced cardiac dysfunction. Crit Care Med 35 (2007) 1599
-
(2007)
Crit Care Med
, vol.35
, pp. 1599
-
-
Rudiger, A.1
Singer, M.2
-
14
-
-
0032566405
-
Inhibitory effects of antioxidants on neonatal rat cardiac myocyte hypertrophy induced by tumor necrosis factor-α and angiotensin II
-
Nakamura K., Fushimi K., Kouchi H., et al. Inhibitory effects of antioxidants on neonatal rat cardiac myocyte hypertrophy induced by tumor necrosis factor-α and angiotensin II. Circulation 98 (1998) 794
-
(1998)
Circulation
, vol.98
, pp. 794
-
-
Nakamura, K.1
Fushimi, K.2
Kouchi, H.3
-
15
-
-
13244256816
-
Heme oxygenase-1: Redox regulation of a stress protein in lung and cell culture models
-
Ryter S.W., and Choi A.M. Heme oxygenase-1: Redox regulation of a stress protein in lung and cell culture models. Antioxid Redox Signal 7 (2005) 80
-
(2005)
Antioxid Redox Signal
, vol.7
, pp. 80
-
-
Ryter, S.W.1
Choi, A.M.2
-
16
-
-
0030188587
-
Heme oxygenase-1: Function, regulation, and implication of a novel stress-inducible protein in oxidant-induced lung injury
-
Choi A.M., and Alam J. Heme oxygenase-1: Function, regulation, and implication of a novel stress-inducible protein in oxidant-induced lung injury. Am J Respir Cell Mol Biol 15 (1996) 9
-
(1996)
Am J Respir Cell Mol Biol
, vol.15
, pp. 9
-
-
Choi, A.M.1
Alam, J.2
-
17
-
-
0032878565
-
Heme oxygenase: Recent advances in understanding its regulation and role
-
Elbirt K.K., and Bonkovsky H.L. Heme oxygenase: Recent advances in understanding its regulation and role. Proc Assoc Am Physicians 111 (1999) 438
-
(1999)
Proc Assoc Am Physicians
, vol.111
, pp. 438
-
-
Elbirt, K.K.1
Bonkovsky, H.L.2
-
18
-
-
13244256816
-
Heme oxygenase-1: Redox regulation of a stress protein in lung and cell culture models
-
Ryter S.W., and Choi A.M. Heme oxygenase-1: Redox regulation of a stress protein in lung and cell culture models. Antioxid Redox Signal 7 (2005) 80
-
(2005)
Antioxid Redox Signal
, vol.7
, pp. 80
-
-
Ryter, S.W.1
Choi, A.M.2
-
19
-
-
0034668216
-
Gene regulation of heme oxygenase-1 as a therapeutic target
-
Immenschuch S., and Ramadori G. Gene regulation of heme oxygenase-1 as a therapeutic target. Biochem Pharmacol 60 (2000) 1121
-
(2000)
Biochem Pharmacol
, vol.60
, pp. 1121
-
-
Immenschuch, S.1
Ramadori, G.2
-
20
-
-
0034694905
-
Induction of heme oxygenase-1 can act protectively against cardiac ischemia/reperfusion in vivo
-
Hangaishi M., Ishizaka N., Aizawa T., et al. Induction of heme oxygenase-1 can act protectively against cardiac ischemia/reperfusion in vivo. Biochem Biophys Res Commun 279 (2000) 582
-
(2000)
Biochem Biophys Res Commun
, vol.279
, pp. 582
-
-
Hangaishi, M.1
Ishizaka, N.2
Aizawa, T.3
-
21
-
-
0035957249
-
H(mox-1) constitutes an adaptive response to effect antioxidant cardioprotection: A study with transgenic mice heterozygous for targeted disruption of the heme oxygenase-1 gene
-
Yoshida T., Maulik N., Ho Y.S., et al. H(mox-1) constitutes an adaptive response to effect antioxidant cardioprotection: A study with transgenic mice heterozygous for targeted disruption of the heme oxygenase-1 gene. Circulation 103 (2001) 1695
-
(2001)
Circulation
, vol.103
, pp. 1695
-
-
Yoshida, T.1
Maulik, N.2
Ho, Y.S.3
-
22
-
-
0035920256
-
Cardiac-specific expression of heme oxygenase-1 protects against ischemia and reperfusion injury in transgenic mice
-
Yet S.F., Tian R., Layne M.D., et al. Cardiac-specific expression of heme oxygenase-1 protects against ischemia and reperfusion injury in transgenic mice. Circ Res 89 (2001) 168
-
(2001)
Circ Res
, vol.89
, pp. 168
-
-
Yet, S.F.1
Tian, R.2
Layne, M.D.3
-
23
-
-
0034053730
-
Heme oxygenase-1 derived bilirubin ameliorates postischemic myocardial dysfunction
-
Clark J.E., Foresti R., Sarathchandra P., et al. Heme oxygenase-1 derived bilirubin ameliorates postischemic myocardial dysfunction. Am J Physiol Heart Circ Physiol 278 (2000) H643
-
(2000)
Am J Physiol Heart Circ Physiol
, vol.278
-
-
Clark, J.E.1
Foresti, R.2
Sarathchandra, P.3
-
24
-
-
0036074215
-
Intestinal preconditioning prevents systemic inflammatory response in hemorrhagic shock. Role of HO-1
-
Tamion F., Richard V., Lacoume Y., et al. Intestinal preconditioning prevents systemic inflammatory response in hemorrhagic shock. Role of HO-1. Am J Physiol Gastrointest Liver Physiol 283 (2002) G408
-
(2002)
Am J Physiol Gastrointest Liver Physiol
, vol.283
-
-
Tamion, F.1
Richard, V.2
Lacoume, Y.3
-
25
-
-
0035920256
-
Cardiac-specific expression of heme oxygenase-1 protects against ischemia and reperfusion injury in transgenic mice
-
Yet S.F., Tian R., Layne M.D., et al. Cardiac-specific expression of heme oxygenase-1 protects against ischemia and reperfusion injury in transgenic mice. Circ Res 89 (2001) 168
-
(2001)
Circ Res
, vol.89
, pp. 168
-
-
Yet, S.F.1
Tian, R.2
Layne, M.D.3
-
26
-
-
0034053730
-
Heme oxygenase-1-derived bilirubin ameliorates postischemic myocardial dysfunction
-
Clark J.E., Foresti R., Sarathchandra P., et al. Heme oxygenase-1-derived bilirubin ameliorates postischemic myocardial dysfunction. Am J Physiol Heart Circ Physiol 278 (2000) H643
-
(2000)
Am J Physiol Heart Circ Physiol
, vol.278
-
-
Clark, J.E.1
Foresti, R.2
Sarathchandra, P.3
-
27
-
-
28744455858
-
Hemin prevents cardiac and diaphragm mitochondrial dysfunction in sepsis
-
Supinski G.S., and Callahan L.A. Hemin prevents cardiac and diaphragm mitochondrial dysfunction in sepsis. Free Radic Biol Med 40 (2006) 127
-
(2006)
Free Radic Biol Med
, vol.40
, pp. 127
-
-
Supinski, G.S.1
Callahan, L.A.2
-
28
-
-
20844461656
-
Allopurinol attenuates left ventricular remodeling and dysfunction after experimental myocardial infarction: A new action for an old drug?
-
Engberding N., Spiekermann S., Schaefer A., et al. Allopurinol attenuates left ventricular remodeling and dysfunction after experimental myocardial infarction: A new action for an old drug?. Circulation 110 (2004) 2175
-
(2004)
Circulation
, vol.110
, pp. 2175
-
-
Engberding, N.1
Spiekermann, S.2
Schaefer, A.3
-
29
-
-
33646679144
-
Overexpression of mitochondrial peroxiredoxin-3 prevents left ventricular remodeling and failure after myocardial infarction in mice
-
Matsushima S., Ide T., Yamato M., et al. Overexpression of mitochondrial peroxiredoxin-3 prevents left ventricular remodeling and failure after myocardial infarction in mice. Circulation 113 (2003) 1779
-
(2003)
Circulation
, vol.113
, pp. 1779
-
-
Matsushima, S.1
Ide, T.2
Yamato, M.3
-
30
-
-
33745202365
-
NADPH oxidase-dependent redox signaling in cardiac hypertrophy, remodeling, and failure
-
Murdoch C.E., Zhang M., Cave A.C., et al. NADPH oxidase-dependent redox signaling in cardiac hypertrophy, remodeling, and failure. Cardiovasc Res 71 (2006) 208
-
(2006)
Cardiovasc Res
, vol.71
, pp. 208
-
-
Murdoch, C.E.1
Zhang, M.2
Cave, A.C.3
-
31
-
-
0034283407
-
Treatment with dimethylthiourea prevents left ventricular remodeling and failure after experimental myocardial infarction in mice: Role of oxidative stress
-
Kinugawa S., Tsutsui H., and Hayashidani S. Treatment with dimethylthiourea prevents left ventricular remodeling and failure after experimental myocardial infarction in mice: Role of oxidative stress. Circ Res 87 (2000) 392
-
(2000)
Circ Res
, vol.87
, pp. 392
-
-
Kinugawa, S.1
Tsutsui, H.2
Hayashidani, S.3
-
32
-
-
14644442283
-
Oxygen, oxidative stress, hypoxia, and heart failure
-
Giordano F.J. Oxygen, oxidative stress, hypoxia, and heart failure. J Investig 115 (2005) 500
-
(2005)
J Investig
, vol.115
, pp. 500
-
-
Giordano, F.J.1
-
33
-
-
0035896205
-
Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarction
-
Ide T., Tsutsui H., Hayashidani S., et al. Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarction. Circ Res 88 (2001) 529
-
(2001)
Circ Res
, vol.88
, pp. 529
-
-
Ide, T.1
Tsutsui, H.2
Hayashidani, S.3
-
34
-
-
0036892344
-
Clinical review: Myocardial depression in sepsis and septic shock
-
Court O., Kumar A., Parrillo J.E., et al. Clinical review: Myocardial depression in sepsis and septic shock. Crit Care 6 (2002) 500
-
(2002)
Crit Care
, vol.6
, pp. 500
-
-
Court, O.1
Kumar, A.2
Parrillo, J.E.3
-
35
-
-
0037307184
-
Overexpression of tumor necrosis factor-α increases production of hydroxyl radical in murine myocardium
-
Machida Y., Kubota T., Kawamura N., et al. Overexpression of tumor necrosis factor-α increases production of hydroxyl radical in murine myocardium. Am J Physiol Heart Circ Physiol 284 (2003) H449
-
(2003)
Am J Physiol Heart Circ Physiol
, vol.284
-
-
Machida, Y.1
Kubota, T.2
Kawamura, N.3
-
36
-
-
0034959968
-
Overexpression of tumor necrosis factor-α activates both anti- and pro-apoptotic pathways in the myocardium
-
Kubota T., Miyagishima M., Frye C.S., et al. Overexpression of tumor necrosis factor-α activates both anti- and pro-apoptotic pathways in the myocardium. J Mol Cell Cardiol 33 (2001) 1331
-
(2001)
J Mol Cell Cardiol
, vol.33
, pp. 1331
-
-
Kubota, T.1
Miyagishima, M.2
Frye, C.S.3
-
37
-
-
0037453028
-
Oxidative stress mediates tumor necrosis factor-α-induced mitochondrial DNA damage and dysfunction in cardiac myocytes
-
Suematsu N., Tsutsui H., Wen J., et al. Oxidative stress mediates tumor necrosis factor-α-induced mitochondrial DNA damage and dysfunction in cardiac myocytes. Circulation 107 (2003) 1418
-
(2003)
Circulation
, vol.107
, pp. 1418
-
-
Suematsu, N.1
Tsutsui, H.2
Wen, J.3
-
38
-
-
0034969250
-
Rapid reactive oxygen species production by mitochondria in endothelial cells exposed to tumor necrosis factor-alpha is mediated by ceramide
-
Corda S., Laplace C., Vicaut E., et al. Rapid reactive oxygen species production by mitochondria in endothelial cells exposed to tumor necrosis factor-alpha is mediated by ceramide. Am J Respir Cell Mol Biol 24 (2001) 762
-
(2001)
Am J Respir Cell Mol Biol
, vol.24
, pp. 762
-
-
Corda, S.1
Laplace, C.2
Vicaut, E.3
-
39
-
-
0031040328
-
Sphingosine mediates the immediate negative inotropic effects of tumor necrosis factor-α in the adult mammalian cardiac myocyte
-
Oral H., Dorn G.W., and Mann D.L. Sphingosine mediates the immediate negative inotropic effects of tumor necrosis factor-α in the adult mammalian cardiac myocyte. J Biol Chem 272 (1997) 4836
-
(1997)
J Biol Chem
, vol.272
, pp. 4836
-
-
Oral, H.1
Dorn, G.W.2
Mann, D.L.3
-
40
-
-
4744348381
-
In vivo TNFα inhibition ameliorates cardiac mitochondrial dysfunction, oxidative stress, and apoptosis in experimental heart failure
-
Moe G.W., Marin-Garcia J., Konig A., et al. In vivo TNFα inhibition ameliorates cardiac mitochondrial dysfunction, oxidative stress, and apoptosis in experimental heart failure. Am J Physiol Heart Circ Physiol 287 (2004) H1813
-
(2004)
Am J Physiol Heart Circ Physiol
, vol.287
-
-
Moe, G.W.1
Marin-Garcia, J.2
Konig, A.3
-
41
-
-
2942555358
-
Heme oxygenase-1: A novel therapeutic target in oxidative tissue injuries
-
Takahashi T., Morita K., Akagi R., et al. Heme oxygenase-1: A novel therapeutic target in oxidative tissue injuries. Curr Med Chem 11 (2004) 1545
-
(2004)
Curr Med Chem
, vol.11
, pp. 1545
-
-
Takahashi, T.1
Morita, K.2
Akagi, R.3
-
42
-
-
0036074215
-
Intestinal preconditioning prevents systemic inflammatory response in hemorrhagic shock. Role of HO-1
-
Tamion F., Richard V., Lacoume Y., et al. Intestinal preconditioning prevents systemic inflammatory response in hemorrhagic shock. Role of HO-1. Am J Physiol Gastrointest Liver Physiol 283 (2002) G408
-
(2002)
Am J Physiol Gastrointest Liver Physiol
, vol.283
-
-
Tamion, F.1
Richard, V.2
Lacoume, Y.3
-
43
-
-
0034053730
-
Heme oxygenase-1-derived bilirubin ameliorates postischemic myocardial dysfunction
-
Clark J.E., Foresti R., Sarathchandra P., et al. Heme oxygenase-1-derived bilirubin ameliorates postischemic myocardial dysfunction. Am J Physiol Heart Circ Physiol 278 (2000) H643
-
(2000)
Am J Physiol Heart Circ Physiol
, vol.278
-
-
Clark, J.E.1
Foresti, R.2
Sarathchandra, P.3
-
44
-
-
0034694905
-
Induction of heme oxygenase-1 can act protectively against cardiac ischemia/reperfusion in vivo
-
Hangaishi M., Ishizaka N., Aizawa T., et al. Induction of heme oxygenase-1 can act protectively against cardiac ischemia/reperfusion in vivo. Biochem Biophys Res Commun 279 (2000) 582
-
(2000)
Biochem Biophys Res Commun
, vol.279
, pp. 582
-
-
Hangaishi, M.1
Ishizaka, N.2
Aizawa, T.3
-
45
-
-
0034642320
-
Endotoxin-induced mortality is related to increased oxidative stress and end-organ dysfunction, not refractory hypotension, in heme oxygenase-1-deficient mice
-
Wiesel P., Patel A.P., DiFonzo N., et al. Endotoxin-induced mortality is related to increased oxidative stress and end-organ dysfunction, not refractory hypotension, in heme oxygenase-1-deficient mice. Circulation 102 (2000) 3015
-
(2000)
Circulation
, vol.102
, pp. 3015
-
-
Wiesel, P.1
Patel, A.P.2
DiFonzo, N.3
-
46
-
-
34247546562
-
Restoring Homeostasis: Is heme oxygénase-1 ready for the clinic?
-
Scott J.R., Chin B.Y., Bilban M.H., et al. Restoring Homeostasis: Is heme oxygénase-1 ready for the clinic?. Trends Pharmacol Sci 28 (2007) 200
-
(2007)
Trends Pharmacol Sci
, vol.28
, pp. 200
-
-
Scott, J.R.1
Chin, B.Y.2
Bilban, M.H.3
-
48
-
-
27944467221
-
Biliverdin administration protects against endotoxin-induced acute lung injury in rats
-
Sarady-Andrews J.K., Liu F., Gallo D., et al. Biliverdin administration protects against endotoxin-induced acute lung injury in rats. Am J Physiol Lung Cell Mol Physiol 289 (2005) L1131
-
(2005)
Am J Physiol Lung Cell Mol Physiol
, vol.289
-
-
Sarady-Andrews, J.K.1
Liu, F.2
Gallo, D.3
|