BCR-ABL stimulates mutagenic homologous DNA double-strand break repair via the DNA-end-processing factor CtIP

Carcinogenesis

Volumn 32, Issue 1, 2011, Pages 27-34

  Salles, Daniela ^a,b,c   Mencalha, Andre L ^a,b   Ireno, Ivanildce C ^c   Wiesmüller, Lisa ^c   Abdelhay, Eliana ^a,b  

^a FEDERAL UNIVERSITY OF RIO DE JANEIRO   (Brazil)

^b INSTITUTO NACIONAL DE CÂNCER   (Brazil)

^c UNIVERSITY OF ULM   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

BCR ABL PROTEIN; CTIP PROTEIN; DEOXYRIBONUCLEOPROTEIN; IMATINIB; UNCLASSIFIED DRUG; CARRIER PROTEIN; NUCLEAR PROTEIN; RBBP8 PROTEIN, HUMAN;

APOPTOSIS; ARTICLE; CELL CYCLE; CHRONIC MYELOID LEUKEMIA; DNA REPAIR; DOUBLE STRANDED DNA BREAK; GENE REPRESSION; HUMAN; HUMAN CELL; HUMAN CELL CULTURE; IMMUNOBLOTTING; IRRADIATION; LEUKEMIA CELL LINE; MUTAGENIC ACTIVITY; PRIORITY JOURNAL; PROTEIN EXPRESSION; UPREGULATION; CELL LINE; CELL TRANSFORMATION; FLUORESCENT ANTIBODY TECHNIQUE; GENETICS; METABOLISM;

CARRIER PROTEINS; CELL LINE; CELL TRANSFORMATION, NEOPLASTIC; DNA BREAKS, DOUBLE-STRANDED; DNA REPAIR; FLUORESCENT ANTIBODY TECHNIQUE; FUSION PROTEINS, BCR-ABL; HUMANS; IMMUNOBLOTTING; LEUKEMIA, MYELOGENOUS, CHRONIC, BCR-ABL POSITIVE; NUCLEAR PROTEINS;

EID: 79952115095     PISSN: 01433334     EISSN: 14602180     Source Type: Journal    
DOI: 10.1093/carcin/bgq216     Document Type: Article

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