The herpes simplex virus type 1 latency-associated transcript can protect neuron-derived C1300 and neuro2A cells from granzyme B-induced apoptosis and CD8 T-cell killing

Journal of Virology

Volumn 85, Issue 5, 2011, Pages 2325-2332

  Jiang, Xianzhi ^a   Chentoufi, Aziz Alami ^a   Hsiang, Chinhui ^a   Carpenter, Dale ^a   Osorio, Nelson ^a   BenMohamed, Lbachir ^a   Fraser, Nigel W ^b   Jones, Clinton ^c   Wechsler, Steven L ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF PENNSYLVANIA   (United States)

^c UNIVERSITY OF NEBRASKA LINCOLN   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; CELL EXTRACT; GRANZYME B; LATENCY ASSOCIATED PEPTIDE;

ANIMAL CELL; APOPTOSIS; ARTICLE; CD8+ T LYMPHOCYTE; CELL FREE SYSTEM; CELL KILLING; CONTROLLED STUDY; CYTOTOXICITY; ENZYME ACTIVATION; GENE EXPRESSION; GENETIC TRANSCRIPTION; HERPES SIMPLEX VIRUS 1; HYPOTHESIS; IN VITRO STUDY; IN VIVO STUDY; INNATE IMMUNITY; MOUSE; NERVE CELL CULTURE; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN CLEAVAGE;

ANIMALS; APOPTOSIS; CD8-POSITIVE T-LYMPHOCYTES; CELL LINE; CYTOTOXICITY, IMMUNOLOGIC; GRANZYMES; HERPES SIMPLEX; HERPESVIRUS 1, HUMAN; HUMANS; MICE; MICE, INBRED C57BL; MICRORNAS; NEURONS; VIRUS ACTIVATION;

HUMAN HERPESVIRUS 1;

EID: 79551719108     PISSN: 0022538X     EISSN: 10985514     Source Type: Journal    
DOI: 10.1128/JVI.01791-10     Document Type: Article

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