Synaptotoxicity of Alzheimer beta amyloid can be explained by its membrane perforating property

PLoS ONE

Volumn 5, Issue 7, 2010, Pages

  Sepulveda, Fernando J ^a   Parodi, Jorge ^a   Peoples, Robert W ^b   Opazo, Carlos ^a   Aguayo, Luis G ^a  

^a UNIVERSITY OF CONCEPCIÓN   (Chile)

^b MARQUETTE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; CALCIUM; ETHIDIUM BROMIDE; GRAMICIDIN; PEPTIDE NA13; PEPTIDE NA15; PEPTIDE NA4A; PEPTIDE NA7; PROTEIN INHIBITOR; UNCLASSIFIED DRUG; PEPTIDE;

ALZHEIMER DISEASE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CALCIUM CELL LEVEL; CALCIUM TRANSPORT; CONTROLLED STUDY; DRUG MECHANISM; EMBRYO; FEMALE; HIPPOCAMPUS; MOUSE; NERVE CELL MEMBRANE; NERVE CELL MEMBRANE CONDUCTANCE; NEUROTOXICITY; NONHUMAN; PROTEIN TARGETING; RAT; SYNAPTIC MEMBRANE; TREATMENT OUTCOME; ANIMAL; C57BL MOUSE; CELL CULTURE; CELL MEMBRANE; DRUG EFFECT; HUMAN; IMMUNOHISTOCHEMISTRY; METABOLISM; NERVE CELL; PREGNANCY; SPRAGUE DAWLEY RAT; WESTERN BLOTTING;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ANIMALS; BLOTTING, WESTERN; CALCIUM; CELL MEMBRANE; CELLS, CULTURED; FEMALE; HUMANS; IMMUNOHISTOCHEMISTRY; MICE; MICE, INBRED C57BL; NEURONS; PEPTIDES; PREGNANCY; RATS; RATS, SPRAGUE-DAWLEY;

EID: 77955701756     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0011820     Document Type: Article

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