Apoptosis and non-inflammatory phagocytosis can be induced by mitochondrial damage without caspases

Cell Death and Differentiation

Volumn 17, Issue 5, 2010, Pages 821-832

  Van Delft, M F ^a,b,c   Smith, D P ^a   Lahoud, M H ^a   Huang, D C S ^a   Adams, J M ^a  

^a WALTER AND ELIZA HALL INSTITUTE OF MEDICAL RESEARCH   (Australia)

^b UNIVERSITY OF MELBOURNE   (Australia)

^c UNIVERSITY HEALTH NETWORK   (Canada)

Author keywords

Apoptosis; Bcl 2 family; Caspases; Mitochondrial membrane potential; Phosphatidylserine

Indexed keywords

ANTINUCLEAR ANTIBODY; CASPASE 9; PHOSPHATIDYLSERINE;

ANIMAL CELL; ANIMAL CELL CULTURE; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; EMBRYO; ENZYME ACTIVITY; FIBROBLAST; FLOW CYTOMETRY; FLUORESCENCE ACTIVATED CELL SORTING; GAMMA RADIATION; HISTOLOGY; IMMUNOFLUORESCENCE; INFLAMMATION; MITOCHONDRIAL MEMBRANE POTENTIAL; MOUSE; NONHUMAN; PHAGOCYTE; PHAGOCYTOSIS; PRIORITY JOURNAL; PROTEIN DEGRADATION; THYMOCYTE; THYMUS;

ANIMALS; APOPTOSIS; CASPASE 9; CASPASES; CELL LINE; CELLS, CULTURED; FLOW CYTOMETRY; FLUORESCENT ANTIBODY TECHNIQUE, INDIRECT; GAMMA RAYS; MEMBRANE POTENTIAL, MITOCHONDRIAL; MICE; MICE, MUTANT STRAINS; MITOCHONDRIA; PHAGOCYTOSIS; THYMUS GLAND;

MUS; VERTEBRATA;

EID: 77950859902     PISSN: 13509047     EISSN: 14765403     Source Type: Journal    
DOI: 10.1038/cdd.2009.166     Document Type: Article

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