Systemic administration of catalpol prevents d-galactose induced mitochondrial dysfunction in mice

Neuroscience Letters

Volumn 473, Issue 3, 2010, Pages 224-228

  Zhang, Xiuli ^a,b   Liu, Weidong ^a   Niu, Xinhua ^a   An, Lijia ^b  

^a BINZHOU MEDICAL UNIVERSITY   (China)

^b DALIAN UNIVERSITY OF TECHNOLOGY   (China)

Author keywords

Aging; Catalpol; Mitochondria; Mitochondrial membrane potential; Reactive oxygen species; Respiratory complex

Indexed keywords

CATALPOL; GALACTOSE; MATRIX METALLOPROTEINASE; NITRIC OXIDE SYNTHASE; PIRACETAM; REACTIVE OXYGEN METABOLITE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; BRAIN MITOCHONDRION; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DRUG MECHANISM; ENZYME ACTIVITY; FEMALE; HIPPOCAMPUS; MALE; MITOCHONDRIAL MEMBRANE POTENTIAL; MOUSE; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; RESPIRATORY CHAIN; TREATMENT DURATION;

ANIMALS; CEREBRAL CORTEX; ELECTRON TRANSPORT COMPLEX I; ELECTRON TRANSPORT COMPLEX IV; FEMALE; GALACTOSE; GLUCOSIDES; HIPPOCAMPUS; IRIDOIDS; MALE; MEMBRANE POTENTIAL, MITOCHONDRIAL; MICE; MITOCHONDRIA; NEUROPROTECTIVE AGENTS; NITRIC OXIDE SYNTHASE; REACTIVE OXYGEN SPECIES;

EID: 77950341945     PISSN: 03043940     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neulet.2010.02.054     Document Type: Article

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