Noxa is necessary for hydrogen peroxide-induced caspase-dependent cell death

FEBS Letters

Volumn 584, Issue 4, 2010, Pages 681-688

  Aikawa, Tomonori ^a   Shinzawa, Koei ^a   Tanaka, Nobuyuki ^b   Tsujimoto, Yoshihide ^a  

^a OSAKA UNIVERSITY MEDICAL SCHOOL   (Japan)

^b NIPPON MEDICAL SCHOOL   (Japan)

Author keywords

Activating transcription factor 4; Apoptosis; Bcl 2; CAMP response element; Hydrogen peroxide; Noxa

Indexed keywords

CASPASE; CYTOCHROME C; HYDROGEN PEROXIDE; MESSENGER RNA; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2; PROTEIN NOXA;

APOPTOSIS; ARTICLE; CELL PROTECTION; CELLULAR DISTRIBUTION; CONTROLLED STUDY; GENE FUNCTION; GENE IDENTIFICATION; GENE OVEREXPRESSION; GENE SILENCING; HUMAN; HUMAN CELL; LEUKEMIA CELL LINE; PRIORITY JOURNAL; PROTEIN DEGRADATION; TRANSCRIPTION INITIATION;

ACTIVATING TRANSCRIPTION FACTOR 4; AMINO ACID CHLOROMETHYL KETONES; APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; BLOTTING, WESTERN; CASPASES; CYSTEINE PROTEINASE INHIBITORS; CYTOCHROMES C; HELA CELLS; HUMANS; HYDROGEN PEROXIDE; JURKAT CELLS; LUCIFERASES; OXIDANTS; PROMOTER REGIONS, GENETIC; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTO-ONCOGENE PROTEINS C-BCL-2; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; RNA INTERFERENCE; TRANSCRIPTIONAL ACTIVATION;

EID: 77649271422     PISSN: 00145793     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.febslet.2010.01.026     Document Type: Article

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