A single dose of passive immunotherapy has extended benefits on synapses and neurites in an Alzheimer's disease mouse model

Brain Research

Volumn 1280, Issue , 2009, Pages 178-185

  Rozkalne, Anete ^a   Spires Jones, Tara L ^a   Stern, Edward A ^a,b   Hyman, Bradley T ^a  

^a HARVARD MEDICAL SCHOOL   (United States)

^b BAR ILAN UNIVERSITY   (Israel)

Author keywords

Alzheimer's; Amyloid; Immunotherapy; Senile plaque; Synaptic plasticity

Indexed keywords

AMYLOID BETA PROTEIN ANTIBODY; ANTIBODY; TAU ANTIBODY; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; BRAIN NERVE CELL; CELL DENSITY; CELL STRUCTURE; CENTRAL NERVOUS SYSTEM; CONTROLLED STUDY; IMMUNOTHERAPY; MOUSE; NERVE CELL PLASTICITY; NEURITE; NONHUMAN; PRIORITY JOURNAL; SENILE PLAQUE; SYNAPSE; THERAPY EFFECT;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; AUTOANTIBODIES; AXONS; BRAIN; DISEASE MODELS, ANIMAL; HUMANS; IMMUNIZATION, PASSIVE; MICE; MICE, TRANSGENIC; NEURONS; RECEPTORS, CELL SURFACE; SENILE PLAQUES; SYNAPSES;

EID: 67649427845     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.brainres.2009.05.045     Document Type: Article

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