Adenovirus expressing Fas ligand gene decreases airway hyper-responsiveness and eosinophilia in a murine model of asthma

Gene Therapy

Volumn 11, Issue 20, 2004, Pages 1497-1505

  Chuang, Y H ^a   Fu, C L ^a   Lo, Y C ^a,b   Chiang, B L ^a  

^a NATIONAL TAIWAN UNIVERSITY   (Taiwan)

^b Taipei Municipal Chung Hsin Hospital   (Taiwan)

Author keywords

Adenoviral vector; Apoptosis; Eosinophils; Eotaxin; IL 13; IL 5

Indexed keywords

ADENOVIRUS VECTOR; CYTOKINE; EOTAXIN; FAS ANTIGEN; FAS LIGAND; INTERLEUKIN 13; INTERLEUKIN 5; OVALBUMIN; TUMOR NECROSIS FACTOR; TUMOR NECROSIS FACTOR ALPHA;

ALLERGIC ASTHMA; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRONCHUS HYPERREACTIVITY; CELL INFILTRATION; CONTROLLED STUDY; EFFECTOR CELL; EOSINOPHILIA; FEMALE; IMMUNIZATION; LYMPHOCYTIC INFILTRATION; MOUSE; NONHUMAN; PRIORITY JOURNAL; RECEPTOR BINDING; RESPIRATORY TRACT INFLAMMATION; SENSITIZATION; TH2 CELL; VIRUS RECOMBINANT;

ADENOVIRIDAE; ANIMALS; ASTHMA; BRONCHIAL HYPERREACTIVITY; BRONCHOALVEOLAR LAVAGE FLUID; DENDRITIC CELLS; FAS LIGAND PROTEIN; FEMALE; GENE THERAPY; GENETIC VECTORS; LEUKOCYTE COUNT; MEMBRANE GLYCOPROTEINS; MICE; MICE, INBRED BALB C; MODELS, ANIMAL; RESPIRATORY MUCOSA; TRANSDUCTION, GENETIC;

ADENOVIRIDAE; MURINAE;

EID: 5444276007     PISSN: 09697128     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.gt.3302325     Document Type: Article

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