-
1
-
-
0028943734
-
Apoptosis in the pathogenesis and treatment of disease
-
Thompson C.B. Apoptosis in the pathogenesis and treatment of disease. Science 267 (1995) 1456-1462
-
(1995)
Science
, vol.267
, pp. 1456-1462
-
-
Thompson, C.B.1
-
2
-
-
0035318470
-
Apoptosis in autoimmune diseases
-
Eguchi K. Apoptosis in autoimmune diseases. Intern. Med. 40 (2001) 275-284
-
(2001)
Intern. Med.
, vol.40
, pp. 275-284
-
-
Eguchi, K.1
-
3
-
-
0242636280
-
Fas-beyond death: a regenerative role for Fas in the nervous system
-
Lambert C., et al. Fas-beyond death: a regenerative role for Fas in the nervous system. Apoptosis 8 (2003) 551-562
-
(2003)
Apoptosis
, vol.8
, pp. 551-562
-
-
Lambert, C.1
-
4
-
-
0037299282
-
Non-apoptotic Fas signaling
-
Wajant H., et al. Non-apoptotic Fas signaling. Cytokine Growth Factor Rev. 14 (2003) 53-66
-
(2003)
Cytokine Growth Factor Rev.
, vol.14
, pp. 53-66
-
-
Wajant, H.1
-
5
-
-
1642575170
-
Fas ligand/Fas system in the brain: regulator of immune and apoptotic responses
-
Choi C., and Benveniste E.N. Fas ligand/Fas system in the brain: regulator of immune and apoptotic responses. Brain Res. Brain Res. Rev. 44 (2004) 65-81
-
(2004)
Brain Res. Brain Res. Rev.
, vol.44
, pp. 65-81
-
-
Choi, C.1
Benveniste, E.N.2
-
6
-
-
0035800225
-
Increased expression of Fas (CD95/APO-1) in adult rat brain after kainate-induced seizures
-
Tan Z., et al. Increased expression of Fas (CD95/APO-1) in adult rat brain after kainate-induced seizures. Neuroreport 12 (2001) 1979-1982
-
(2001)
Neuroreport
, vol.12
, pp. 1979-1982
-
-
Tan, Z.1
-
7
-
-
3042845128
-
The role of Fas-mediated apoptosis after traumatic spinal cord injury
-
Yoshino O., et al. The role of Fas-mediated apoptosis after traumatic spinal cord injury. Spine 29 (2004) 1394-1404
-
(2004)
Spine
, vol.29
, pp. 1394-1404
-
-
Yoshino, O.1
-
8
-
-
11144354396
-
Neutralization of CD95 ligand promotes regeneration and functional recovery after spinal cord injury
-
Demjen D., et al. Neutralization of CD95 ligand promotes regeneration and functional recovery after spinal cord injury. Nat. Med. 10 (2004) 389-395
-
(2004)
Nat. Med.
, vol.10
, pp. 389-395
-
-
Demjen, D.1
-
9
-
-
27744542561
-
FAS deficiency reduces apoptosis, spares axons and improves function after spinal cord injury
-
Casha S., et al. FAS deficiency reduces apoptosis, spares axons and improves function after spinal cord injury. Exp. Neurol. 196 (2005) 390-400
-
(2005)
Exp. Neurol.
, vol.196
, pp. 390-400
-
-
Casha, S.1
-
10
-
-
33646575557
-
Inhibition of Fas-mediated apoptosis through administration of soluble Fas receptor improves functional outcome and reduces posttraumatic axonal degeneration after acute spinal cord injury
-
Ackery A., et al. Inhibition of Fas-mediated apoptosis through administration of soluble Fas receptor improves functional outcome and reduces posttraumatic axonal degeneration after acute spinal cord injury. J. Neurotrauma 23 (2006) 604-616
-
(2006)
J. Neurotrauma
, vol.23
, pp. 604-616
-
-
Ackery, A.1
-
11
-
-
34547909939
-
CD95/Fas mediates cognitive improvement after traumatic brain injury
-
Beier C.P., et al. CD95/Fas mediates cognitive improvement after traumatic brain injury. Cell Res. 17 (2007) 732-734
-
(2007)
Cell Res.
, vol.17
, pp. 732-734
-
-
Beier, C.P.1
-
12
-
-
0031252369
-
Fas and Fas ligand enhance the pathogenesis of experimental allergic encephalomyelitis, but are not essential for immune privilege in the central nervous system
-
Sabelko K.A., et al. Fas and Fas ligand enhance the pathogenesis of experimental allergic encephalomyelitis, but are not essential for immune privilege in the central nervous system. J. Immunol. 159 (1997) 3096-3099
-
(1997)
J. Immunol.
, vol.159
, pp. 3096-3099
-
-
Sabelko, K.A.1
-
13
-
-
0031253737
-
Fas- and FasL-deficient mice are resistant to induction of autoimmune encephalomyelitis
-
Waldner H., et al. Fas- and FasL-deficient mice are resistant to induction of autoimmune encephalomyelitis. J. Immunol. 159 (1997) 3100-3103
-
(1997)
J. Immunol.
, vol.159
, pp. 3100-3103
-
-
Waldner, H.1
-
14
-
-
0033378856
-
Role of Fas-FasL interactions in the pathogenesis and regulation of autoimmune demyelinating disease
-
Sabelko-Downes K.A., et al. Role of Fas-FasL interactions in the pathogenesis and regulation of autoimmune demyelinating disease. J. Neuroimmunol. 100 (1999) 42-52
-
(1999)
J. Neuroimmunol.
, vol.100
, pp. 42-52
-
-
Sabelko-Downes, K.A.1
-
15
-
-
0033152818
-
Evidence for Fas-dependent and Fas-independent mechanisms in the pathogenesis of experimental autoimmune encephalomyelitis
-
Dittel B.N., et al. Evidence for Fas-dependent and Fas-independent mechanisms in the pathogenesis of experimental autoimmune encephalomyelitis. J. Immunol. 162 (1999) 6392-6400
-
(1999)
J. Immunol.
, vol.162
, pp. 6392-6400
-
-
Dittel, B.N.1
-
16
-
-
27144460509
-
Apoptosis of oligodendrocytes via Fas and TNF-R1 is a key event in the induction of experimental autoimmune encephalomyelitis
-
Hovelmeyer N., et al. Apoptosis of oligodendrocytes via Fas and TNF-R1 is a key event in the induction of experimental autoimmune encephalomyelitis. J. Immunol. 175 (2005) 5875-5884
-
(2005)
J. Immunol.
, vol.175
, pp. 5875-5884
-
-
Hovelmeyer, N.1
-
17
-
-
0033562658
-
CD95 ligand (Fas-L/APO-1L) and tumor necrosis factor-related apoptosis-inducing ligand mediate ischemia-induced apoptosis in neurons
-
Martin-Villalba A., et al. CD95 ligand (Fas-L/APO-1L) and tumor necrosis factor-related apoptosis-inducing ligand mediate ischemia-induced apoptosis in neurons. J. Neurosci. 19 (1999) 3809-3817
-
(1999)
J. Neurosci.
, vol.19
, pp. 3809-3817
-
-
Martin-Villalba, A.1
-
18
-
-
0034666684
-
Fas (CD95/APO-1) plays a role in the pathophysiology of focal cerebral ischemia
-
Rosenbaum D.M., et al. Fas (CD95/APO-1) plays a role in the pathophysiology of focal cerebral ischemia. J. Neurosci. Res. 61 (2000) 686-692
-
(2000)
J. Neurosci. Res.
, vol.61
, pp. 686-692
-
-
Rosenbaum, D.M.1
-
19
-
-
0034915133
-
Therapeutic neutralization of CD95-ligand and TNF attenuates brain damage in stroke
-
Martin-Villalba A., et al. Therapeutic neutralization of CD95-ligand and TNF attenuates brain damage in stroke. Cell Death Differ. 8 (2001) 679-686
-
(2001)
Cell Death Differ.
, vol.8
, pp. 679-686
-
-
Martin-Villalba, A.1
-
20
-
-
4444377604
-
Neonatal mice lacking functional Fas death receptors are resistant to hypoxic-ischemic brain injury
-
Graham E.M., et al. Neonatal mice lacking functional Fas death receptors are resistant to hypoxic-ischemic brain injury. Neurobiol. Dis. 17 (2004) 89-98
-
(2004)
Neurobiol. Dis.
, vol.17
, pp. 89-98
-
-
Graham, E.M.1
-
21
-
-
0141457901
-
Fas/tumor necrosis factor receptor death signaling is required for axotomy-induced death of motoneurons in vivo
-
Ugolini G., et al. Fas/tumor necrosis factor receptor death signaling is required for axotomy-induced death of motoneurons in vivo. J. Neurosci. 23 (2003) 8526-8531
-
(2003)
J. Neurosci.
, vol.23
, pp. 8526-8531
-
-
Ugolini, G.1
-
22
-
-
21844470751
-
Adult motor neuron apoptosis is mediated by nitric oxide and Fas death receptor linked by DNA damage and p53 activation
-
Martin L.J., et al. Adult motor neuron apoptosis is mediated by nitric oxide and Fas death receptor linked by DNA damage and p53 activation. J. Neurosci. 25 (2005) 6449-6459
-
(2005)
J. Neurosci.
, vol.25
, pp. 6449-6459
-
-
Martin, L.J.1
-
23
-
-
0033615957
-
Programmed cell death of embryonic motoneurons triggered through the Fas death receptor
-
Raoul C., et al. Programmed cell death of embryonic motoneurons triggered through the Fas death receptor. J. Cell Biol. 147 (1999) 1049-1062
-
(1999)
J. Cell Biol.
, vol.147
, pp. 1049-1062
-
-
Raoul, C.1
-
24
-
-
33645822300
-
Chronic activation in presymptomatic amyotrophic lateral sclerosis (ALS) mice of a feedback loop involving Fas, Daxx, and FasL
-
Raoul C., et al. Chronic activation in presymptomatic amyotrophic lateral sclerosis (ALS) mice of a feedback loop involving Fas, Daxx, and FasL. Proc. Natl. Acad. Sci. U. S. A. 103 (2006) 6007-6012
-
(2006)
Proc. Natl. Acad. Sci. U. S. A.
, vol.103
, pp. 6007-6012
-
-
Raoul, C.1
-
25
-
-
33751256823
-
Loss of Fas ligand-function improves survival in G93A-transgenic ALS mice
-
Petri S., et al. Loss of Fas ligand-function improves survival in G93A-transgenic ALS mice. J. Neurol. Sci. 251 (2006) 44-49
-
(2006)
J. Neurol. Sci.
, vol.251
, pp. 44-49
-
-
Petri, S.1
-
26
-
-
34547764229
-
Fas small interfering RNA reduces motoneuron death in amyotrophic lateral sclerosis mice
-
Locatelli F., et al. Fas small interfering RNA reduces motoneuron death in amyotrophic lateral sclerosis mice. Ann. Neurol. 62 (2007) 81-92
-
(2007)
Ann. Neurol.
, vol.62
, pp. 81-92
-
-
Locatelli, F.1
-
27
-
-
0035478618
-
β-Amyloid induces neuronal apoptosis via a mechanism that involves the c-Jun N-terminal kinase pathway and the induction of Fas ligand
-
Morishima Y., et al. β-Amyloid induces neuronal apoptosis via a mechanism that involves the c-Jun N-terminal kinase pathway and the induction of Fas ligand. J. Neurosci. 21 (2001) 7551-7560
-
(2001)
J. Neurosci.
, vol.21
, pp. 7551-7560
-
-
Morishima, Y.1
-
28
-
-
0038079348
-
Fas and Fas ligand are associated with neuritic degeneration in the AD brain and participate in β-amyloid-induced neuronal death
-
Su J.H., et al. Fas and Fas ligand are associated with neuritic degeneration in the AD brain and participate in β-amyloid-induced neuronal death. Neurobiol. Dis. 12 (2003) 182-193
-
(2003)
Neurobiol. Dis.
, vol.12
, pp. 182-193
-
-
Su, J.H.1
-
29
-
-
0030897321
-
Delayed neurite regeneration and its improvement by nerve growth factor (NGF) in dorsal root ganglia from MRL-lpr/lpr mice in vitro
-
Hikawa N., et al. Delayed neurite regeneration and its improvement by nerve growth factor (NGF) in dorsal root ganglia from MRL-lpr/lpr mice in vitro. J. Neurol. Sci. 149 (1997) 13-17
-
(1997)
J. Neurol. Sci.
, vol.149
, pp. 13-17
-
-
Hikawa, N.1
-
30
-
-
0037321075
-
Fas engagement induces neurite growth through ERK activation and p35 upregulation
-
Desbarats J., et al. Fas engagement induces neurite growth through ERK activation and p35 upregulation. Nat. Cell Biol. 5 (2003) 118-125
-
(2003)
Nat. Cell Biol.
, vol.5
, pp. 118-125
-
-
Desbarats, J.1
-
31
-
-
33645006173
-
Control of neuronal branching by the death receptor CD95 (Fas/Apo-1)
-
Zuliani C., et al. Control of neuronal branching by the death receptor CD95 (Fas/Apo-1). Cell Death Differ. 13 (2006) 31-40
-
(2006)
Cell Death Differ.
, vol.13
, pp. 31-40
-
-
Zuliani, C.1
-
32
-
-
0032125972
-
Progressive atrophy of pyramidal neuron dendrites in autoimmune MRL-lpr mice
-
Sakic B., et al. Progressive atrophy of pyramidal neuron dendrites in autoimmune MRL-lpr mice. J. Neuroimmunol. 87 (1998) 162-170
-
(1998)
J. Neuroimmunol.
, vol.87
, pp. 162-170
-
-
Sakic, B.1
-
33
-
-
10744223673
-
Regulation of dopaminergic loss by Fas in a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease
-
Hayley S., et al. Regulation of dopaminergic loss by Fas in a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease. J. Neurosci. 24 (2004) 2045-2053
-
(2004)
J. Neurosci.
, vol.24
, pp. 2045-2053
-
-
Hayley, S.1
-
34
-
-
24344455580
-
Defective Fas expression exacerbates neurotoxicity in a model of Parkinson's disease
-
Landau A.M., et al. Defective Fas expression exacerbates neurotoxicity in a model of Parkinson's disease. J. Exp. Med. 202 (2005) 575-581
-
(2005)
J. Exp. Med.
, vol.202
, pp. 575-581
-
-
Landau, A.M.1
-
35
-
-
2542449195
-
T cell-specific ablation of Fas leads to Fas ligand-mediated lymphocyte depletion and inflammatory pulmonary fibrosis
-
Hao Z., et al. T cell-specific ablation of Fas leads to Fas ligand-mediated lymphocyte depletion and inflammatory pulmonary fibrosis. J. Exp. Med. 199 (2004) 1355-1365
-
(2004)
J. Exp. Med.
, vol.199
, pp. 1355-1365
-
-
Hao, Z.1
-
36
-
-
0033835065
-
Fas engagement accelerates liver regeneration after partial hepatectomy
-
Desbarats J., and Newell M.K. Fas engagement accelerates liver regeneration after partial hepatectomy. Nat. Med. 6 (2000) 920-923
-
(2000)
Nat. Med.
, vol.6
, pp. 920-923
-
-
Desbarats, J.1
Newell, M.K.2
-
37
-
-
34247536682
-
The CD95 receptor: apoptosis revisited
-
Peter M.E., et al. The CD95 receptor: apoptosis revisited. Cell 129 (2007) 447-450
-
(2007)
Cell
, vol.129
, pp. 447-450
-
-
Peter, M.E.1
-
38
-
-
0035844129
-
MAPK/ERK overrides the apoptotic signaling from Fas, TNF, and TRAIL receptors
-
Tran S.E., et al. MAPK/ERK overrides the apoptotic signaling from Fas, TNF, and TRAIL receptors. J. Biol. Chem. 276 (2001) 16484-16490
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 16484-16490
-
-
Tran, S.E.1
-
39
-
-
27544432516
-
Nonapoptotic functions of FADD-binding death receptors and their signaling molecules
-
Park S.M., et al. Nonapoptotic functions of FADD-binding death receptors and their signaling molecules. Curr. Opin. Cell Biol. 17 (2005) 610-616
-
(2005)
Curr. Opin. Cell Biol.
, vol.17
, pp. 610-616
-
-
Park, S.M.1
-
40
-
-
19344372267
-
Does CD95 have tumor promoting activities?
-
Peter M.E., et al. Does CD95 have tumor promoting activities?. Biochim. Biophys. Acta 1755 (2005) 25-36
-
(2005)
Biochim. Biophys. Acta
, vol.1755
, pp. 25-36
-
-
Peter, M.E.1
-
41
-
-
40149099894
-
Yes and PI3K bind CD95 to signal invasion of glioblastoma
-
Kleber S., et al. Yes and PI3K bind CD95 to signal invasion of glioblastoma. Cancer Cell 13 (2008) 235-248
-
(2008)
Cancer Cell
, vol.13
, pp. 235-248
-
-
Kleber, S.1
-
42
-
-
50249118029
-
A novel juxtamembrane domain in TNF receptor superfamily molecules activates Rac1 and controls neurite growth
-
10.1091/mbc.E08-02-0161
-
Ruan W., et al. A novel juxtamembrane domain in TNF receptor superfamily molecules activates Rac1 and controls neurite growth. Mol. Biol. Cell (2008). http://www.molbiolcell.org 10.1091/mbc.E08-02-0161
-
(2008)
Mol. Biol. Cell
-
-
Ruan, W.1
-
43
-
-
9244249814
-
Absence of caspase 8 and high expression of PED protect primitive neural cells from cell death
-
Ricci-Vitiani L., et al. Absence of caspase 8 and high expression of PED protect primitive neural cells from cell death. J. Exp. Med. 200 (2004) 1257-1266
-
(2004)
J. Exp. Med.
, vol.200
, pp. 1257-1266
-
-
Ricci-Vitiani, L.1
-
44
-
-
1842767239
-
Neural stem cells and cell death
-
Ceccatelli S., et al. Neural stem cells and cell death. Toxicol. Lett. 149 (2004) 59-66
-
(2004)
Toxicol. Lett.
, vol.149
, pp. 59-66
-
-
Ceccatelli, S.1
-
45
-
-
42949162823
-
A new class of reverse signaling costimulators belongs to the TNF family
-
Sun M., and Fink P.J. A new class of reverse signaling costimulators belongs to the TNF family. J. Immunol. 179 (2007) 4307-4312
-
(2007)
J. Immunol.
, vol.179
, pp. 4307-4312
-
-
Sun, M.1
Fink, P.J.2
-
46
-
-
0037276437
-
The CD95(APO-1/Fas) DISC and beyond
-
Peter M.E., and Krammer P.H. The CD95(APO-1/Fas) DISC and beyond. Cell Death Differ. 10 (2003) 26-35
-
(2003)
Cell Death Differ.
, vol.10
, pp. 26-35
-
-
Peter, M.E.1
Krammer, P.H.2
-
47
-
-
39049148397
-
Cellular FLICE-like inhibitory protein (C-FLIP): a novel target for cancer therapy
-
Safa A.R., et al. Cellular FLICE-like inhibitory protein (C-FLIP): a novel target for cancer therapy. Curr. Cancer Drug Targets 8 (2008) 37-46
-
(2008)
Curr. Cancer Drug Targets
, vol.8
, pp. 37-46
-
-
Safa, A.R.1
-
48
-
-
1642386039
-
N-terminal fragment of c-FLIP(L) processed by caspase 8 specifically interacts with TRAF2 and induces activation of the NF-κB signaling pathway
-
Kataoka T., and Tschopp J. N-terminal fragment of c-FLIP(L) processed by caspase 8 specifically interacts with TRAF2 and induces activation of the NF-κB signaling pathway. Mol. Cell. Biol. 24 (2004) 2627-2636
-
(2004)
Mol. Cell. Biol.
, vol.24
, pp. 2627-2636
-
-
Kataoka, T.1
Tschopp, J.2
-
49
-
-
34548295607
-
Antioxidant status alters levels of Fas-associated death domain-like IL-1B-converting enzyme inhibitory protein following neonatal hypoxia-ischemia
-
Payton K.S., et al. Antioxidant status alters levels of Fas-associated death domain-like IL-1B-converting enzyme inhibitory protein following neonatal hypoxia-ischemia. Dev. Neurosci. 29 (2007) 403-411
-
(2007)
Dev. Neurosci.
, vol.29
, pp. 403-411
-
-
Payton, K.S.1
-
50
-
-
34250799663
-
FLIP(L) protects neurons against in vivo ischemia and in vitro glucose deprivation-induced cell death
-
Taoufik E., et al. FLIP(L) protects neurons against in vivo ischemia and in vitro glucose deprivation-induced cell death. J. Neurosci. 27 (2007) 6633-6646
-
(2007)
J. Neurosci.
, vol.27
, pp. 6633-6646
-
-
Taoufik, E.1
-
51
-
-
2942696314
-
The expression of PEA-15 (phosphoprotein enriched in astrocytes of 15 kDa) defines subpopulations of astrocytes and neurons throughout the adult mouse brain
-
Sharif A., et al. The expression of PEA-15 (phosphoprotein enriched in astrocytes of 15 kDa) defines subpopulations of astrocytes and neurons throughout the adult mouse brain. Neuroscience 126 (2004) 263-275
-
(2004)
Neuroscience
, vol.126
, pp. 263-275
-
-
Sharif, A.1
-
52
-
-
0035117038
-
Induction and intracellular regulation of tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) mediated apoptosis in human malignant glioma cells
-
Hao C., et al. Induction and intracellular regulation of tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) mediated apoptosis in human malignant glioma cells. Cancer Res. 61 (2001) 1162-1170
-
(2001)
Cancer Res.
, vol.61
, pp. 1162-1170
-
-
Hao, C.1
-
53
-
-
0035929583
-
Involvement of phospholipase D in sphingosine 1-phosphate-induced activation of phosphatidylinositol 3-kinase and Akt in Chinese hamster ovary cells overexpressing EDG3
-
Banno Y., et al. Involvement of phospholipase D in sphingosine 1-phosphate-induced activation of phosphatidylinositol 3-kinase and Akt in Chinese hamster ovary cells overexpressing EDG3. J. Biol. Chem. 276 (2001) 35622-35628
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 35622-35628
-
-
Banno, Y.1
-
54
-
-
39149129327
-
The PEA-15/PED protein protects glioblastoma cells from glucose deprivation-induced apoptosis via the ERK/MAP kinase pathway
-
Eckert A., et al. The PEA-15/PED protein protects glioblastoma cells from glucose deprivation-induced apoptosis via the ERK/MAP kinase pathway. Oncogene 27 (2008) 1155-1166
-
(2008)
Oncogene
, vol.27
, pp. 1155-1166
-
-
Eckert, A.1
-
55
-
-
25844522837
-
Phosphorylation of PEA-15 switches its binding specificity from ERK/MAPK to FADD
-
Renganathan H., et al. Phosphorylation of PEA-15 switches its binding specificity from ERK/MAPK to FADD. Biochem. J. 390 (2005) 729-735
-
(2005)
Biochem. J.
, vol.390
, pp. 729-735
-
-
Renganathan, H.1
-
56
-
-
35448996570
-
The long form of Fas apoptotic inhibitory molecule is expressed specifically in neurons and protects them against death receptor-triggered apoptosis
-
Segura M.F., et al. The long form of Fas apoptotic inhibitory molecule is expressed specifically in neurons and protects them against death receptor-triggered apoptosis. J. Neurosci. 27 (2007) 11228-11241
-
(2007)
J. Neurosci.
, vol.27
, pp. 11228-11241
-
-
Segura, M.F.1
-
57
-
-
8444221220
-
The death receptor antagonist FAIM promotes neurite outgrowth by a mechanism that depends on ERK and NF-κ B signaling
-
Sole C., et al. The death receptor antagonist FAIM promotes neurite outgrowth by a mechanism that depends on ERK and NF-κ B signaling. J. Cell Biol. 167 (2004) 479-492
-
(2004)
J. Cell Biol.
, vol.167
, pp. 479-492
-
-
Sole, C.1
-
58
-
-
0141656235
-
Constitutive expression of p55TNFR mRNA and mitogen-specific up-regulation of TNF α and p75TNFR mRNA in mouse brain
-
Bette M., et al. Constitutive expression of p55TNFR mRNA and mitogen-specific up-regulation of TNF α and p75TNFR mRNA in mouse brain. J. Comp. Neurol. 465 (2003) 417-430
-
(2003)
J. Comp. Neurol.
, vol.465
, pp. 417-430
-
-
Bette, M.1
-
59
-
-
0033607280
-
LFG: an anti-apoptotic gene that provides protection from Fas-mediated cell death
-
Somia N.V., et al. LFG: an anti-apoptotic gene that provides protection from Fas-mediated cell death. Proc. Natl. Acad. Sci. U. S. A. 96 (1999) 12667-12672
-
(1999)
Proc. Natl. Acad. Sci. U. S. A.
, vol.96
, pp. 12667-12672
-
-
Somia, N.V.1
-
60
-
-
22544433438
-
FasL (CD95L/APO-1L) resistance of neurons mediated by phosphatidylinositol 3-kinase-Akt/protein kinase B-dependent expression of lifeguard/neuronal membrane protein 35
-
Beier C.P., et al. FasL (CD95L/APO-1L) resistance of neurons mediated by phosphatidylinositol 3-kinase-Akt/protein kinase B-dependent expression of lifeguard/neuronal membrane protein 35. J. Neurosci. 25 (2005) 6765-6774
-
(2005)
J. Neurosci.
, vol.25
, pp. 6765-6774
-
-
Beier, C.P.1
-
61
-
-
34548624517
-
Lifeguard/neuronal membrane protein 35 regulates Fas ligand-mediated apoptosis in neurons via microdomain recruitment
-
Fernandez M., et al. Lifeguard/neuronal membrane protein 35 regulates Fas ligand-mediated apoptosis in neurons via microdomain recruitment. J. Neurochem. 103 (2007) 190-203
-
(2007)
J. Neurochem.
, vol.103
, pp. 190-203
-
-
Fernandez, M.1
-
62
-
-
0031865211
-
Neural membrane protein 35 (NMP35): a novel member of a gene family which is highly expressed in the adult nervous system
-
Schweitzer B., et al. Neural membrane protein 35 (NMP35): a novel member of a gene family which is highly expressed in the adult nervous system. Mol. Cell. Neurosci. 11 (1998) 260-273
-
(1998)
Mol. Cell. Neurosci.
, vol.11
, pp. 260-273
-
-
Schweitzer, B.1
-
63
-
-
0037202282
-
Neural membrane protein 35/Lifeguard is localized at postsynaptic sites and in dendrites.
-
Schweitzer B., et al. Neural membrane protein 35/Lifeguard is localized at postsynaptic sites and in dendrites. Brain Res. Mol. Brain Res. 107 (2002) 47-56
-
(2002)
Brain Res. Mol. Brain Res.
, vol.107
, pp. 47-56
-
-
Schweitzer, B.1
-
64
-
-
3442895056
-
CD95 death-inducing signaling complex formation and internalization occur in lipid rafts of type I and type II cells
-
Eramo A., et al. CD95 death-inducing signaling complex formation and internalization occur in lipid rafts of type I and type II cells. Eur. J. Immunol. 34 (2004) 1930-1940
-
(2004)
Eur. J. Immunol.
, vol.34
, pp. 1930-1940
-
-
Eramo, A.1
-
65
-
-
34249100534
-
FasL, Fas, and death-inducing signaling complex (DISC) proteins are recruited to membrane rafts after spinal cord injury
-
Davis A.R., et al. FasL, Fas, and death-inducing signaling complex (DISC) proteins are recruited to membrane rafts after spinal cord injury. J. Neurotrauma 24 (2007) 823-834
-
(2007)
J. Neurotrauma
, vol.24
, pp. 823-834
-
-
Davis, A.R.1
-
66
-
-
26844507890
-
FAS associated phosphatase (FAP-1) blocks apoptosis of astrocytomas through dephosphorylation of FAS
-
Foehr E.D., et al. FAS associated phosphatase (FAP-1) blocks apoptosis of astrocytomas through dephosphorylation of FAS. J. Neurooncol. 74 (2005) 241-248
-
(2005)
J. Neurooncol.
, vol.74
, pp. 241-248
-
-
Foehr, E.D.1
-
67
-
-
2442593499
-
Therapeutic potential of inhibiting Bruton's tyrosine kinase (BTK)
-
Vassilev A.O., and Uckun F.M. Therapeutic potential of inhibiting Bruton's tyrosine kinase (BTK). Curr. Pharm. Des. 10 (2004) 1757-1766
-
(2004)
Curr. Pharm. Des.
, vol.10
, pp. 1757-1766
-
-
Vassilev, A.O.1
Uckun, F.M.2
-
68
-
-
15744374877
-
The mouse cell surface protein TOSO regulates Fas/Fas ligand-induced apoptosis through its binding to Fas-associated death domain
-
Song Y., and Jacob C.O. The mouse cell surface protein TOSO regulates Fas/Fas ligand-induced apoptosis through its binding to Fas-associated death domain. J. Biol. Chem. 280 (2005) 9618-9626
-
(2005)
J. Biol. Chem.
, vol.280
, pp. 9618-9626
-
-
Song, Y.1
Jacob, C.O.2
-
69
-
-
6344276922
-
Instant decisions: transcription-independent control of death-receptor-mediated apoptosis
-
Tran S.E., et al. Instant decisions: transcription-independent control of death-receptor-mediated apoptosis. Trends Biochem. Sci. 29 (2004) 601-608
-
(2004)
Trends Biochem. Sci.
, vol.29
, pp. 601-608
-
-
Tran, S.E.1
-
70
-
-
0035957426
-
Erythropoietin prevents neuronal apoptosis after cerebral ischemia and metabolic stress
-
Siren A.L., et al. Erythropoietin prevents neuronal apoptosis after cerebral ischemia and metabolic stress. Proc. Natl. Acad. Sci. U. S. A. 98 (2001) 4044-4049
-
(2001)
Proc. Natl. Acad. Sci. U. S. A.
, vol.98
, pp. 4044-4049
-
-
Siren, A.L.1
|