The effect of CAG repeat length polymorphism in the murine glucocorticoid receptor on transactivation potential

Experimental and Molecular Pathology

Volumn 84, Issue 3, 2008, Pages 200-205

  Yee, Kristen S ^a   Cho, Kiho ^a   Green, Tajia ^a   Chandler, John ^a   Greenhalgh, David G ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Burn injury; CAG repeat; CD14 knockout mouse; Glucocorticoid; Glucocorticoid receptor; Inflammation; Injury response; Sepsis

Indexed keywords

BETA ACTIN; GENOMIC DNA; GLUCOCORTICOID RECEPTOR; LUCIFERASE; TRINUCLEOTIDE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BURN; CONTROLLED STUDY; DNA POLYMORPHISM; DNA PURIFICATION; FEMALE; GENE SEQUENCE; GENETIC ENGINEERING; GENETIC TRANSCRIPTION; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; POLYMERASE CHAIN REACTION; PROTEIN EXPRESSION; SEPSIS; SIGNAL TRANSDUCTION; TRANSACTIVATION; TRINUCLEOTIDE REPEAT; WESTERN BLOTTING;

ANIMALS; ANTIGENS, CD14; BURNS; FEMALE; GENE EXPRESSION REGULATION; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; POLYMORPHISM, GENETIC; RECEPTORS, GLUCOCORTICOID; RESPONSE ELEMENTS; TRANS-ACTIVATION (GENETICS); TRINUCLEOTIDE REPEATS;

MURINAE; MUS;

EID: 45849083124     PISSN: 00144800     EISSN: 10960945     Source Type: Journal    
DOI: 10.1016/j.yexmp.2008.04.003     Document Type: Article

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