UVA-induced cell cycle progression is mediated by a disintegrin and metalloprotease/epidermal growth factor receptor/AKT/cyclin D1 pathways in keratinocytes

Cancer Research

Volumn 68, Issue 10, 2008, Pages 3752-3758

  He, Yu Ying ^a,b   Council, Sarah E ^a,c   Feng, Li ^a   Chignell, Colin F ^a  

^a NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES   (United States)

^b UNIVERSITY OF CHICAGO   (United States)

^c UNIVERSITY OF NORTH CAROLINA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIN D1; DISINTEGRIN; EPIDERMAL GROWTH FACTOR RECEPTOR; EPIDERMAL GROWTH FACTOR RECEPTOR KINASE INHIBITOR; ILOMASTAT; METALLOPROTEINASE; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE INHIBITOR; PHOSPHATIDYLINOSITOL KINASE; PROTEIN KINASE B; SMALL INTERFERING RNA; TUMOR NECROSIS FACTOR ALPHA CONVERTING ENZYME; ADAM PROTEIN; ANTINEOPLASTIC AGENT; ENZYME INHIBITOR; PHOSPHATIDYLINOSITOL 3 KINASE; TUMOR NECROSIS FACTOR-ALPHA CONVERTASE;

ARTICLE; CELL CYCLE PROGRESSION; CELL CYCLE S PHASE; CELL PROLIFERATION; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME INHIBITION; HUMAN; HUMAN CELL; KERATINOCYTE; PRIORITY JOURNAL; SKIN CANCER; ULTRAVIOLET A RADIATION; BIOLOGICAL MODEL; CELL CYCLE; DRUG EFFECT; METABOLISM; PATHOLOGY; SKIN TUMOR; TUMOR CELL LINE; ULTRAVIOLET RADIATION;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ADAM PROTEINS; ANTINEOPLASTIC AGENTS; CELL CYCLE; CELL LINE, TUMOR; CELL PROLIFERATION; CYCLIN D1; ENZYME INHIBITORS; HUMANS; KERATINOCYTES; MODELS, BIOLOGICAL; PROTO-ONCOGENE PROTEINS C-AKT; RECEPTOR, EPIDERMAL GROWTH FACTOR; SKIN NEOPLASMS; ULTRAVIOLET RAYS;

EID: 45549084638     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: 10.1158/0008-5472.CAN-07-6138     Document Type: Article

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