Increase in bax expression and apoptosis are associated in Huntington's disease progression

Neuroscience Letters

Volumn 438, Issue 1, 2008, Pages 59-63

  Teles, A V F F ^a   Rosenstock, T R ^a   Okuno, C S ^a   Lopes, G S ^a   Bertoncini, C R A ^b   Smaili, S S ^a  

^a FEDERAL UNIVERSITY OF SÃO PAULO   (Brazil)

^b UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

Aging; Apoptosis; bax; bcl 2; Cell death; Huntington's disease; mRNA; Neurodegeneration

Indexed keywords

PROTEIN BAX; PROTEIN BCL 2;

ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; DISEASE ASSOCIATION; DISEASE COURSE; HUNTINGTON CHOREA; MALE; MOUSE; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION;

ANIMALS; APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CELL NUCLEUS; CEREBRAL CORTEX; DISEASE PROGRESSION; GENE EXPRESSION; GENETIC PREDISPOSITION TO DISEASE; HUNTINGTON DISEASE; IN SITU NICK-END LABELING; MALE; MICE; MICE, INBRED C57BL; MICE, INBRED CBA; MICE, TRANSGENIC; NERVE DEGENERATION; NEURONS; PROTO-ONCOGENE PROTEINS C-BCL-2; RNA, MESSENGER; UP-REGULATION;

EID: 43549102381     PISSN: 03043940     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neulet.2008.03.062     Document Type: Article

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