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Volumn 234-235, Issue , 2002, Pages 119-124
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Doxorubicin-induced apoptosis: Implications in cardiotoxicity
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Author keywords
Apoptosis; Caspase activation; Doxorubicin; Endothelial nitric oxide synthase; Reactive oxygen species
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Indexed keywords
DOXORUBICIN;
HYDROGEN PEROXIDE;
IRON;
NITRIC OXIDE SYNTHASE;
REACTIVE OXYGEN METABOLITE;
CHELATING AGENT;
RAZOXANE;
ANIMAL;
APOPTOSIS;
CARDIOMYOPATHY;
CHEMICALLY INDUCED DISORDER;
DRUG EFFECT;
GENETICS;
HUMAN;
METABOLISM;
PATHOLOGY;
REVIEW;
SIGNAL TRANSDUCTION;
ANIMAL EXPERIMENT;
ARTICLE;
BREAST CANCER;
CANCER CHEMOTHERAPY;
CANCER PATIENT;
CELL DEATH;
CONCENTRATION RESPONSE;
DRUG EXPOSURE;
DRUG INHIBITION;
ENDOTHELIUM CELL;
FEMALE;
GENETIC TRANSCRIPTION;
HEART MUSCLE CELL;
IRON CHELATION;
NONHUMAN;
OXIDATION REDUCTION REACTION;
OXIDATIVE STRESS;
PROTEIN EXPRESSION;
ANIMALS;
APOPTOSIS;
CARDIOMYOPATHIES;
DOXORUBICIN;
HUMANS;
HYDROGEN PEROXIDE;
IRON;
NITRIC OXIDE SYNTHASE;
REACTIVE OXYGEN SPECIES;
SIGNAL TRANSDUCTION;
ANIMAL;
HUMAN;
MYOCARDIAL DISEASES;
NITRIC-OXIDE SYNTHASE;
SUPPORT, U.S. GOV'T, P.H.S.;
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EID: 4243907381
PISSN: 03008177
EISSN: None
Source Type: Journal
DOI: 10.1023/A:1015976430790 Document Type: Article |
Times cited : (366)
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References (60)
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