H2AX phosphorylation after UV irradiation is triggered by DNA repair intermediates and is mediated by the ATR kinase

Carcinogenesis

Volumn 28, Issue 11, 2007, Pages 2298-2304

  Hanasoge, Sheela ^a   Ljungman, Mats ^a  

^a UNIVERSITY OF MICHIGAN   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ATM PROTEIN; ATR PROTEIN; DOUBLE STRANDED DNA; HISTONE H2AX; PHOSPHATIDYLINOSITOL 3 KINASE INHIBITOR;

ARTICLE; ATAXIA TELANGIECTASIA; CELL CYCLE G1 PHASE; CELL CYCLE G2 PHASE; CELL CYCLE M PHASE; CONTROLLED STUDY; DNA REPAIR; DNA REPLICATION; DNA STRAND BREAKAGE; DNA SYNTHESIS INHIBITION; FIBROBLAST CULTURE; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; SECKEL SYNDROME; ULTRAVIOLET C RADIATION; XERODERMA PIGMENTOSUM;

BLOTTING, WESTERN; CELL CYCLE; CELL CYCLE PROTEINS; DNA REPAIR; DOSE-RESPONSE RELATIONSHIP, RADIATION; FLOW CYTOMETRY; HISTONES; HUMANS; IMMUNOHISTOCHEMISTRY; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; ULTRAVIOLET RAYS;

EID: 36148951013     PISSN: 01433334     EISSN: 14602180     Source Type: Journal    
DOI: 10.1093/carcin/bgm157     Document Type: Article

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