Suppression of lipopolysaccharide- and tumour necrosis factor-α- induced interleukin (IL)-8 expression by glucocorticoids involves changes in IL-8 promoter acetylation

Clinical and Experimental Immunology

Volumn 150, Issue 1, 2007, Pages 151-157

  Tsaprouni, L G ^a   Ito, K ^a   Adcock, I M ^a   Punchard, N ^b  

^a NATIONAL HEART AND LUNG INSTITUTE   (United Kingdom)

^b UNIVERSITY OF BEDFORDSHIRE   (United Kingdom)

Author keywords

Histone acetylation; Monocytes; T cells; Triamcinolone; Trichostatin A

Indexed keywords

DEXAMETHASONE; GLUCOCORTICOID; INTERLEUKIN 8; LIPOPOLYSACCHARIDE; TRIAMCINOLONE ACETONIDE; TRICHOSTATIN A; TUMOR NECROSIS FACTOR ALPHA;

ACETYLATION; ARTICLE; CONTROLLED STUDY; DRUG INHIBITION; GENE EXPRESSION; HUMAN; HUMAN CELL; IMMUNOPRECIPITATION; LEUKEMIA CELL LINE; LYMPHOCYTE; MACROPHAGE; PRIORITY JOURNAL;

ACETYLATION; DEXAMETHASONE; GENE EXPRESSION REGULATION; GLUCOCORTICOIDS; HISTONES; HUMANS; HYDROXAMIC ACIDS; INTERLEUKIN-8; LIPOPOLYSACCHARIDES; LYMPHOCYTES; MACROPHAGES; PROMOTER REGIONS (GENETICS); TRIAMCINOLONE ACETONIDE; TUMOR CELLS, CULTURED; TUMOR NECROSIS FACTOR-ALPHA;

EID: 34548541573     PISSN: 00099104     EISSN: 13652249     Source Type: Journal    
DOI: 10.1111/j.1365-2249.2007.03484.x     Document Type: Article

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