Giα1-Mediated cardiac electrophysiological remodeling and arrhythmia in hypertrophic cardiomyopathy

Circulation

Volumn 116, Issue 6, 2007, Pages 596-605

  Ruan, Hongmei ^a   Mitchell, Scherise ^a   Vainoriene, Monika ^a   Lou, Qing ^a   Xie, Lai Hua ^a   Ren, Shuxun ^a   Goldhaber, Joshua I ^a   Wang, Yibin ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Arrhythmia; Cardiomyopathy; Electrophysiology; Hypertrophy; Signal transduction

Indexed keywords

CALCIUM; CYCLIC AMP DEPENDENT PROTEIN KINASE; GALPHA INHIBITING SUBUNIT 1; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHOLAMBAN; PROTEIN DERIVATIVE; RAF PROTEIN; RAS PROTEIN; UNCLASSIFIED DRUG; INHIBITORY GUANINE NUCLEOTIDE BINDING PROTEIN;

ACTION POTENTIAL; ANIMAL CELL; ARTICLE; CELL ISOLATION; CONTROLLED STUDY; ENZYME PHOSPHORYLATION; GENE SWITCHING; HEART ARRHYTHMIA; HEART ELECTROPHYSIOLOGY; HEART MUSCLE CELL; HYPERTROPHIC CARDIOMYOPATHY; MOUSE; NONHUMAN; PERTUSSIS; POTASSIUM CURRENT; PRIORITY JOURNAL; PROTEIN EXPRESSION; SARCOPLASMIC RETICULUM; SIGNAL TRANSDUCTION; SODIUM CALCIUM EXCHANGE; ANIMAL; BIOSYNTHESIS; C57BL MOUSE; COMPARATIVE STUDY; ELECTROCARDIOGRAPHY; ELECTROPHYSIOLOGY; GENETICS; HEART VENTRICLE REMODELING; METABOLISM; METHODOLOGY; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; TRANSGENIC MOUSE;

ANIMALS; ARRHYTHMIA; CARDIOMYOPATHY, HYPERTROPHIC; ELECTROCARDIOGRAPHY; ELECTROPHYSIOLOGY; GTP-BINDING PROTEIN ALPHA SUBUNITS, GI-GO; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MYOCYTES, CARDIAC; VENTRICULAR REMODELING;

EID: 34548151001     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/CIRCULATIONAHA.106.682773     Document Type: Article

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