Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 286, Issue 1 55-1, 2004, Pages

  Zheng, Meizi ^a   Dilly, Keith ^a,b   Dos Santos Cruz, Jader ^b   Li, Manxiang ^a   Gu, Yusu ^c   Ursitti, Jeanine A ^b   Chen, Ju ^c   Ross Jr , John ^c   Chien, Kenneth R ^c   Lederer, Jonathan W ^a,b   Wang, Yibin ^a,b,d  

^a UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF MARYLAND BIOTECHNOLOGY INSTITUTE   (United States)

^c UNIVERSITY OF CALIFORNIA SAN DIEGO   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Calcium regulation; Diastolic dysfunction; Phosphatase; Protein phosphorylation

Indexed keywords

ADENOSINE TRIPHOSPHATASE (CALCIUM); CALCIUM CHANNEL L TYPE; DOBUTAMINE; GUANINE NUCLEOTIDE BINDING PROTEIN; MESSENGER RNA; PHOSPHOLAMBAN; PHOSPHOPROTEIN PHOSPHATASE 1; RAS PROTEIN;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CALCIUM SIGNALING; CONTROLLED STUDY; HEART MUSCLE CELL; HEMODYNAMICS; HYPERTROPHIC CARDIOMYOPATHY; IMMUNOBLOTTING; IMMUNOFLUORESCENCE; IMMUNOHISTOCHEMISTRY; MOUSE; NONHUMAN; ONCOGENE H RAS; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; SARCOMERE; SARCOPLASMIC RETICULUM; TRANSGENIC MOUSE;

EID: 9144227436     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00110.2003     Document Type: Article

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