-
2
-
-
0031686409
-
Internal tandem duplication of the FLT3 gene is a novel modality of elongation mutation which causes constitutive activation of the product
-
Kiyoi H, Towatari M, Yokota S, et al. Internal tandem duplication of the FLT3 gene is a novel modality of elongation mutation which causes constitutive activation of the product. Leukemia. 1998;12:1333-1337.
-
(1998)
Leukemia
, vol.12
, pp. 1333-1337
-
-
Kiyoi, H.1
Towatari, M.2
Yokota, S.3
-
3
-
-
0035871889
-
Activating mutation of D835 within the activation loop of FLT3 in human hematologic malignancies
-
Yamamoto Y, Kiyoi H, Nakano Y, et al. Activating mutation of D835 within the activation loop of FLT3 in human hematologic malignancies. Blood. 2001;97:2434-2439.
-
(2001)
Blood
, vol.97
, pp. 2434-2439
-
-
Yamamoto, Y.1
Kiyoi, H.2
Nakano, Y.3
-
4
-
-
0030588487
-
The CBFbeta subunit is essential for CBFalpha2 (AML1) function in vivo
-
Wang Q, Stacy T, Miller JD, et al. The CBFbeta subunit is essential for CBFalpha2 (AML1) function in vivo. Cell. 1996;87:697-708.
-
(1996)
Cell
, vol.87
, pp. 697-708
-
-
Wang, Q.1
Stacy, T.2
Miller, J.D.3
-
5
-
-
0037309377
-
The core-binding factor leukemias: Lessons learned from murine models
-
Downing JR. The core-binding factor leukemias: lessons learned from murine models. Curr Opin Genet Dev. 2003;13:48-54.
-
(2003)
Curr Opin Genet Dev
, vol.13
, pp. 48-54
-
-
Downing, J.R.1
-
6
-
-
33344465478
-
KIT-D816 mutations in AML1-ETO-positive AML are associated with impaired event-free and overall survival
-
Schnittger S, Kohl TM, Haferlach T, et al. KIT-D816 mutations in AML1-ETO-positive AML are associated with impaired event-free and overall survival. Blood. 2006;107:1791-1799.
-
(2006)
Blood
, vol.107
, pp. 1791-1799
-
-
Schnittger, S.1
Kohl, T.M.2
Haferlach, T.3
-
7
-
-
0029019659
-
AML1 and the 8;21 and 3;21 translocations in acute and chronic myeloid leukemia
-
Nucifora G, Rowley JD. AML1 and the 8;21 and 3;21 translocations in acute and chronic myeloid leukemia. Blood. 1995;86:1-14.
-
(1995)
Blood
, vol.86
, pp. 1-14
-
-
Nucifora, G.1
Rowley, J.D.2
-
8
-
-
16944367034
-
ETV6 is the target of chromosome 12p deletions in t(12; 21) childhood acute lymphocytic leukemia
-
Cave H, Cacheux V, Raynaud S, et al. ETV6 is the target of chromosome 12p deletions in t(12; 21) childhood acute lymphocytic leukemia. Leukemia. 1997;11:1459-1464.
-
(1997)
Leukemia
, vol.11
, pp. 1459-1464
-
-
Cave, H.1
Cacheux, V.2
Raynaud, S.3
-
9
-
-
0036463950
-
Expression of a conditional AML1-ETO oncogene bypasses embryonic lethality and establishes a murine model of human t(8;21) acute myeloid leukemia
-
Higuchi M, O'Brien D, Kumaravelu P, et al. Expression of a conditional AML1-ETO oncogene bypasses embryonic lethality and establishes a murine model of human t(8;21) acute myeloid leukemia. Cancer Cell. 2002;1:63-74.
-
(2002)
Cancer Cell
, vol.1
, pp. 63-74
-
-
Higuchi, M.1
O'Brien, D.2
Kumaravelu, P.3
-
10
-
-
23644454231
-
The AML1-ETO fusion gene and the FLT3 length mutation collaborate in inducing acute leukemia in mice
-
Schessl C, Rawat VP, Cusan M, et al. The AML1-ETO fusion gene and the FLT3 length mutation collaborate in inducing acute leukemia in mice. J Clin Invest. 2005;115:2159-2168.
-
(2005)
J Clin Invest
, vol.115
, pp. 2159-2168
-
-
Schessl, C.1
Rawat, V.P.2
Cusan, M.3
-
11
-
-
0033559746
-
Biallelic and heterozygous point mutations in the runt domain of the AML1/PEBP2alphaB gene associated with myeloblastic leukemias
-
Osato M, Asou N, Abdalla E, et al. Biallelic and heterozygous point mutations in the runt domain of the AML1/PEBP2alphaB gene associated with myeloblastic leukemias. Blood. 1999;93:1817-1824.
-
(1999)
Blood
, vol.93
, pp. 1817-1824
-
-
Osato, M.1
Asou, N.2
Abdalla, E.3
-
12
-
-
0037092550
-
Unlike AML1, CBFbeta gene is not deregulated by point mutations in acute myeloid leukemia and in myelodysplastic syndromes
-
Leroy H, Roumier C, Grardel-Duflos N, et al. Unlike AML1, CBFbeta gene is not deregulated by point mutations in acute myeloid leukemia and in myelodysplastic syndromes. Blood. 2002;99:3848-3850.
-
(2002)
Blood
, vol.99
, pp. 3848-3850
-
-
Leroy, H.1
Roumier, C.2
Grardel-Duflos, N.3
-
13
-
-
0032830638
-
Haplo-insufficiency of CBFA2 causes familial thrombocytopenia with propensity to develop acute myelogenous leukaemia
-
Song WJ, Sullivan MG, Legare RD, et al. Haplo-insufficiency of CBFA2 causes familial thrombocytopenia with propensity to develop acute myelogenous leukaemia. Nat Genet. 1999;23:166-175.
-
(1999)
Nat Genet
, vol.23
, pp. 166-175
-
-
Song, W.J.1
Sullivan, M.G.2
Legare, R.D.3
-
14
-
-
0034667690
-
High incidence of biallelic point mutations in the Runt domain of the AML1/PEBP2 alpha B gene in Mo acute myeloid leukemia and in myeloid malignancies with acquired trisomy 21
-
Preudhomme C, Warot-Loze D, Roumier C, et al. High incidence of biallelic point mutations in the Runt domain of the AML1/PEBP2 alpha B gene in Mo acute myeloid leukemia and in myeloid malignancies with acquired trisomy 21. Blood. 2000;96:2862-2869.
-
(2000)
Blood
, vol.96
, pp. 2862-2869
-
-
Preudhomme, C.1
Warot-Loze, D.2
Roumier, C.3
-
15
-
-
0042522486
-
AML1/RUNX1 mutations are infrequent, but related to AML-M0, acquired trisomy 21, and leukemic transformation in pediatric hematologic malignancies
-
Taketani T, Taki T, Takita J, et al. AML1/RUNX1 mutations are infrequent, but related to AML-M0, acquired trisomy 21, and leukemic transformation in pediatric hematologic malignancies. Genes Chromosomes Cancer. 2003;38:1-7.
-
(2003)
Genes Chromosomes Cancer
, vol.38
, pp. 1-7
-
-
Taketani, T.1
Taki, T.2
Takita, J.3
-
16
-
-
9144270483
-
Dual mutations in the AML1 and FLT3 genes are associated with leukemogenesis in acute myeloblastic leukemia of the M0 subtype
-
Matsuno N, Osato M, Yamashita N, et al. Dual mutations in the AML1 and FLT3 genes are associated with leukemogenesis in acute myeloblastic leukemia of the M0 subtype. Leukemia. 2003;17:2492-2499.
-
(2003)
Leukemia
, vol.17
, pp. 2492-2499
-
-
Matsuno, N.1
Osato, M.2
Yamashita, N.3
-
17
-
-
1542373639
-
High incidence of somatic mutations in the AML1/RUNX1 gene in myelodysplastic syndrome and low blast percentage myeloid leukemia with myelodysplasia
-
Harada H, Harada Y, Niimi H, et al. High incidence of somatic mutations in the AML1/RUNX1 gene in myelodysplastic syndrome and low blast percentage myeloid leukemia with myelodysplasia. Blood. 2004;103:2316-2324.
-
(2004)
Blood
, vol.103
, pp. 2316-2324
-
-
Harada, H.1
Harada, Y.2
Niimi, H.3
-
18
-
-
4444302228
-
Mutations of AML1 are common in therapy-related myelodysplasia following therapy with alkylating agents and are significantly associated with deletion or loss of chromosome arm 7q and with subsequent leukemic transformation
-
Christiansen DH, Andersen MK, Pedersen-Bjergaard J. Mutations of AML1 are common in therapy-related myelodysplasia following therapy with alkylating agents and are significantly associated with deletion or loss of chromosome arm 7q and with subsequent leukemic transformation. Blood. 2004;104:1474-1481.
-
(2004)
Blood
, vol.104
, pp. 1474-1481
-
-
Christiansen, D.H.1
Andersen, M.K.2
Pedersen-Bjergaard, J.3
-
19
-
-
33644518397
-
Cooperation of activating Ras/rtk signal transduction pathway mutations and inactivating myeloid differentiation gene mutations in M0 AML: A study of 45 patients
-
Roumier C, Lejeune-Dumoulin S, Renneville A, et al. Cooperation of activating Ras/rtk signal transduction pathway mutations and inactivating myeloid differentiation gene mutations in M0 AML: a study of 45 patients. Leukemia. 2006;20:433-436.
-
(2006)
Leukemia
, vol.20
, pp. 433-436
-
-
Roumier, C.1
Lejeune-Dumoulin, S.2
Renneville, A.3
-
20
-
-
33646475438
-
Hyperactivation of the RAS signaling pathway in myelodysplastic syndrome with AML1/RUNX1 point mutations
-
Niimi H, Harada H, Harada Y, et al. Hyperactivation of the RAS signaling pathway in myelodysplastic syndrome with AML1/RUNX1 point mutations. Leukemia. 2006;20:635-644.
-
(2006)
Leukemia
, vol.20
, pp. 635-644
-
-
Niimi, H.1
Harada, H.2
Harada, Y.3
-
21
-
-
29144497917
-
Detailed analysis of FLT3 expression levels in acute myeloid leukemia
-
Kuchenbauer F, Kern W, Schoch C, et al. Detailed analysis of FLT3 expression levels in acute myeloid leukemia. Haematologica. 2005;90:1617-1625.
-
(2005)
Haematologica
, vol.90
, pp. 1617-1625
-
-
Kuchenbauer, F.1
Kern, W.2
Schoch, C.3
-
22
-
-
10744230464
-
Biologic and clinical significance of the FLT3 transcript level in acute myeloid leukemia
-
Ozeki K, Kiyoi H, Hirose Y, et al. Biologic and clinical significance of the FLT3 transcript level in acute myeloid leukemia. Blood. 2004;103:1901-1908.
-
(2004)
Blood
, vol.103
, pp. 1901-1908
-
-
Ozeki, K.1
Kiyoi, H.2
Hirose, Y.3
-
23
-
-
0013312329
-
Inhibition of FLT3 in MLL: Validation of a therapeutic target identified by gene expression based classi-fication
-
Armstrong SA, Kung AL, Mabon ME, et al. Inhibition of FLT3 in MLL: validation of a therapeutic target identified by gene expression based classi-fication. Cancer Cell. 2003;3:173-183.
-
(2003)
Cancer Cell
, vol.3
, pp. 173-183
-
-
Armstrong, S.A.1
Kung, A.L.2
Mabon, M.E.3
-
24
-
-
0033395790
-
The World Health Organization classification of neoplastic diseases of the hematopoietic and lymphoid tissues: Report of the Clinical Advisory Committee meeting, Airlie House, Virginia, November, 1997
-
Harris NL, Jaffe ES, Diebold J, et al. The World Health Organization classification of neoplastic diseases of the hematopoietic and lymphoid tissues: report of the Clinical Advisory Committee meeting, Airlie House, Virginia, November, 1997. Ann Oncol. 1999;10:1419-1432.
-
(1999)
Ann Oncol
, vol.10
, pp. 1419-1432
-
-
Harris, N.L.1
Jaffe, E.S.2
Diebold, J.3
-
25
-
-
0022135739
-
Proposed revised criteria for the classification of acute myeloid leukemia: A report of the French-American-British Cooperative Group
-
Bennett JM, Catovsky D, Daniel MT, et al. Proposed revised criteria for the classification of acute myeloid leukemia: a report of the French-American-British Cooperative Group. Ann Intern Med. 1985;103:620-625.
-
(1985)
Ann Intern Med
, vol.103
, pp. 620-625
-
-
Bennett, J.M.1
Catovsky, D.2
Daniel, M.T.3
-
26
-
-
0017162163
-
Proposals for the classification of the acute leukaemias: French-American-British (FAB) co-operative group
-
Bennett JM, Catovsky D, Daniel MT, et al. Proposals for the classification of the acute leukaemias: French-American-British (FAB) co-operative group. Br J Haematol. 1976;33:451-458.
-
(1976)
Br J Haematol
, vol.33
, pp. 451-458
-
-
Bennett, J.M.1
Catovsky, D.2
Daniel, M.T.3
-
27
-
-
33751168475
-
Immunostimulatory oligonucleotide-induced metaphase cytogenetics detect chromosomal aberrations in 80% of CLL patients: A study of 132 CLL cases with correlation to FISH, IgVH status, and CD38 expression
-
Dicker F, Schnittger S, Haferlach T, Kern W, Schoch C. Immunostimulatory oligonucleotide-induced metaphase cytogenetics detect chromosomal aberrations in 80% of CLL patients: a study of 132 CLL cases with correlation to FISH, IgVH status, and CD38 expression. Blood. 2006;108:3152-3160.
-
(2006)
Blood
, vol.108
, pp. 3152-3160
-
-
Dicker, F.1
Schnittger, S.2
Haferlach, T.3
Kern, W.4
Schoch, C.5
-
28
-
-
33646575624
-
Implications of NRAS mutations in AML: A study of 2502 patients
-
Bacher U, Haferlach T, Schoch C, Kern W, Schnittger S. Implications of NRAS mutations in AML: a study of 2502 patients. Blood. 2006;107:3847-3853.
-
(2006)
Blood
, vol.107
, pp. 3847-3853
-
-
Bacher, U.1
Haferlach, T.2
Schoch, C.3
Kern, W.4
Schnittger, S.5
-
29
-
-
0036659931
-
Analysis of FLT3 length mutations in 1003 patients with acute myeloid leukemia: Correlation to cytogenetics, FAB subtype, and prognosis in the AMLCG study and usefulness as a marker for the detection of minimal residual disease
-
Schnittger S, Schoch C, Dugas M, et al. Analysis of FLT3 length mutations in 1003 patients with acute myeloid leukemia: correlation to cytogenetics, FAB subtype, and prognosis in the AMLCG study and usefulness as a marker for the detection of minimal residual disease. Blood. 2002;100:59-66.
-
(2002)
Blood
, vol.100
, pp. 59-66
-
-
Schnittger, S.1
Schoch, C.2
Dugas, M.3
-
30
-
-
85009854501
-
Comparison of chromosome banding analysis, interphase-and hypermetaphase-FISH, qualitative and quantitative PCR for diagnosis and for follow-up in chronic myeloid leukemia: A study on 350 cases
-
Schoch C, Schnittger S, Bursch S, et al. Comparison of chromosome banding analysis, interphase-and hypermetaphase-FISH, qualitative and quantitative PCR for diagnosis and for follow-up in chronic myeloid leukemia: a study on 350 cases. Leukemia. 2002;16:53-59.
-
(2002)
Leukemia
, vol.16
, pp. 53-59
-
-
Schoch, C.1
Schnittger, S.2
Bursch, S.3
-
32
-
-
19444370843
-
Acute myeloid leukemia with a complex aberrant karyotype is a distinct biological entity characterized by genomic imbalances and a specific gene expression profile
-
Schoch C, Kern W, Kohlmann A, et al. Acute myeloid leukemia with a complex aberrant karyotype is a distinct biological entity characterized by genomic imbalances and a specific gene expression profile. Genes Chromosomes Cancer. 2005;43:227-238.
-
(2005)
Genes Chromosomes Cancer
, vol.43
, pp. 227-238
-
-
Schoch, C.1
Kern, W.2
Kohlmann, A.3
-
33
-
-
0033987736
-
Mutation nomenclature extensions and suggestions to describe complex mutations: A discussion
-
den Dunnen JT, Antonarakis SE. Mutation nomenclature extensions and suggestions to describe complex mutations: a discussion. Hum Mutat. 2000;15:7-12.
-
(2000)
Hum Mutat
, vol.15
, pp. 7-12
-
-
den Dunnen, J.T.1
Antonarakis, S.E.2
-
34
-
-
0035831044
-
Structural analyses of DNA recognition by the AML1/Runx-1 Runt domain and its allosteric control by CBFbeta
-
Tahirov TH, Inoue-Bungo T, Morii H, et al. Structural analyses of DNA recognition by the AML1/Runx-1 Runt domain and its allosteric control by CBFbeta. Cell. 2001;104:755-767.
-
(2001)
Cell
, vol.104
, pp. 755-767
-
-
Tahirov, T.H.1
Inoue-Bungo, T.2
Morii, H.3
-
35
-
-
0026757174
-
Identification of breakpoints in t(8;21) acute myelogenous leukemia and isolation of a fusion transcript, AML1/ETO, with similarity to Drosophila segmentation gene, runt
-
Erickson P, Gao J, Chang KS, et al. Identification of breakpoints in t(8;21) acute myelogenous leukemia and isolation of a fusion transcript, AML1/ETO, with similarity to Drosophila segmentation gene, runt. Blood. 1992;80:1825-1831.
-
(1992)
Blood
, vol.80
, pp. 1825-1831
-
-
Erickson, P.1
Gao, J.2
Chang, K.S.3
-
36
-
-
0037438508
-
Implications of somatic mutations in the AML1 gene in radiation-associated and therapy-related myelodysplastic syndrome/acute myeloid leukemia
-
Harada H, Harada Y, Tanaka H, Kimura A, Inaba T. Implications of somatic mutations in the AML1 gene in radiation-associated and therapy-related myelodysplastic syndrome/acute myeloid leukemia. Blood. 2003;101:673-680.
-
(2003)
Blood
, vol.101
, pp. 673-680
-
-
Harada, H.1
Harada, Y.2
Tanaka, H.3
Kimura, A.4
Inaba, T.5
-
37
-
-
0032878113
-
Optimal temperature selection for mutation detection by denaturing HPLC and comparison to single-stranded conformation polymorphism and heteroduplex analysis
-
Jones AC, Austin J, Hansen N, et al. Optimal temperature selection for mutation detection by denaturing HPLC and comparison to single-stranded conformation polymorphism and heteroduplex analysis. Clin Chem. 1999;45:1133-1140.
-
(1999)
Clin Chem
, vol.45
, pp. 1133-1140
-
-
Jones, A.C.1
Austin, J.2
Hansen, N.3
-
38
-
-
0025147767
-
Trisomy 13: A new recurring chromosome abnormality in acute leukemia
-
Dohner H, Arthur DC, Ball ED, et al. Trisomy 13: a new recurring chromosome abnormality in acute leukemia. Blood. 1990;76:1614-1621.
-
(1990)
Blood
, vol.76
, pp. 1614-1621
-
-
Dohner, H.1
Arthur, D.C.2
Ball, E.D.3
-
39
-
-
0037114753
-
Pretreatment cytogenetic abnormalities are predictive of induction success, cumulative incidence of relapse, and overall survival in adult patients with de novo acute myeloid leukemia: Results from Cancer and Leukemia Group B (CALGB 8461)
-
Byrd JC, Mrozek K, Dodge RK, et al. Pretreatment cytogenetic abnormalities are predictive of induction success, cumulative incidence of relapse, and overall survival in adult patients with de novo acute myeloid leukemia: results from Cancer and Leukemia Group B (CALGB 8461). Blood. 2002;100:4325-4336.
-
(2002)
Blood
, vol.100
, pp. 4325-4336
-
-
Byrd, J.C.1
Mrozek, K.2
Dodge, R.K.3
-
40
-
-
7444267879
-
Cytogenetic profile in de novo acute myeloid leukemia with FAB subtypes M0, M1, and M2: A study based on 652 cases analyzed with morphology, cytogenetics, and fluorescence in situ hybridization
-
Klaus M, Haferlach T, Schnittger S, et al. Cytogenetic profile in de novo acute myeloid leukemia with FAB subtypes M0, M1, and M2: a study based on 652 cases analyzed with morphology, cytogenetics, and fluorescence in situ hybridization. Cancer Genet Cytogenet. 2004;155:47-56.
-
(2004)
Cancer Genet Cytogenet
, vol.155
, pp. 47-56
-
-
Klaus, M.1
Haferlach, T.2
Schnittger, S.3
-
41
-
-
0036814971
-
Molecular genetics of human leukemias: New insights into therapy
-
Gilliland DG. Molecular genetics of human leukemias: new insights into therapy. Semin Hematol. 2002;39:6-11.
-
(2002)
Semin Hematol
, vol.39
, pp. 6-11
-
-
Gilliland, D.G.1
-
42
-
-
0034101453
-
Trisomy 4 leading to duplication of a mutated KIT allele in acute myeloid leukemia with mast cell involvement
-
Beghini A, Ripamonti CB, Castorina P, et al. Trisomy 4 leading to duplication of a mutated KIT allele in acute myeloid leukemia with mast cell involvement. Cancer Genet Cytogenet. 2000;119:26-31.
-
(2000)
Cancer Genet Cytogenet
, vol.119
, pp. 26-31
-
-
Beghini, A.1
Ripamonti, C.B.2
Castorina, P.3
-
43
-
-
0032514365
-
Duplication and overexpression of the mutant allele of the MET proto-oncogene in multiple hereditary papillary renal cell tumours
-
Fischer J, Palmedo G, von Knobloch R, et al. Duplication and overexpression of the mutant allele of the MET proto-oncogene in multiple hereditary papillary renal cell tumours. Oncogene. 1998;17:733-739.
-
(1998)
Oncogene
, vol.17
, pp. 733-739
-
-
Fischer, J.1
Palmedo, G.2
von Knobloch, R.3
-
44
-
-
17344373892
-
-
Zhuang Z, Park WS, Pack S, et al. Trisomy 7-harbouring non-random duplication of the mutant MET allele in hereditary papillary renal carcinomas. Nat Genet. 1998;20:66-69.
-
Zhuang Z, Park WS, Pack S, et al. Trisomy 7-harbouring non-random duplication of the mutant MET allele in hereditary papillary renal carcinomas. Nat Genet. 1998;20:66-69.
-
-
-
-
45
-
-
19944431253
-
AML1-ETO and C-KIT mutation/ overexpression in t(8;21) leukemia: Implication in stepwise leukemogenesis and response to Gleevec
-
Wang YY, Zhou GB, Yin T, et al. AML1-ETO and C-KIT mutation/ overexpression in t(8;21) leukemia: implication in stepwise leukemogenesis and response to Gleevec. Proc Natl Acad Sci U S A. 2005;102:1104-1109.
-
(2005)
Proc Natl Acad Sci U S A
, vol.102
, pp. 1104-1109
-
-
Wang, Y.Y.1
Zhou, G.B.2
Yin, T.3
-
46
-
-
21744435615
-
Genomic gains and losses influence expression levels of genes located within the affected regions: A study on acute myeloid leukemias with trisomy 8, 11, or 13, monosomy 7, or deletion 5q
-
Schoch C, Kohlmann A, Dugas M, et al. Genomic gains and losses influence expression levels of genes located within the affected regions: a study on acute myeloid leukemias with trisomy 8, 11, or 13, monosomy 7, or deletion 5q. Leukemia. 2005;19:1224-1228.
-
(2005)
Leukemia
, vol.19
, pp. 1224-1228
-
-
Schoch, C.1
Kohlmann, A.2
Dugas, M.3
-
47
-
-
33845240584
-
Phase 1 clinical results with tandutinib (MLN518), a novel FLT3 antagonist, in patients with acute myelogenous leukemia or high-risk myelodysplastic syndrome: Safety, pharmacokinetics, and pharmacodynamics
-
DeAngelo DJ, Stone RM, Heaney ML, et al. Phase 1 clinical results with tandutinib (MLN518), a novel FLT3 antagonist, in patients with acute myelogenous leukemia or high-risk myelodysplastic syndrome: safety, pharmacokinetics, and pharmacodynamics. Blood. 2006;108:3674-3681.
-
(2006)
Blood
, vol.108
, pp. 3674-3681
-
-
DeAngelo, D.J.1
Stone, R.M.2
Heaney, M.L.3
-
48
-
-
33751173636
-
A phase 2 trial of the FLT3 inhibitor lestaurtinib (CEP701) as first-line treatment for older patients with acute myeloid leukemia not considered fit for intensive chemotherapy
-
Knapper S, Burnett AK, Littlewood T, et al. A phase 2 trial of the FLT3 inhibitor lestaurtinib (CEP701) as first-line treatment for older patients with acute myeloid leukemia not considered fit for intensive chemotherapy. Blood. 2006;108:3262-3270.
-
(2006)
Blood
, vol.108
, pp. 3262-3270
-
-
Knapper, S.1
Burnett, A.K.2
Littlewood, T.3
|