Bistability in apoptosis: Roles of Bax, Bcl-2, and mitochondrial permeability transition pores

Biophysical Journal

Volumn 90, Issue 5, 2006, Pages 1546-1559

  Bagci, E Z ^a   Vodovotz, Y ^a,b   Billiar, T R ^a   Ermentrout, G B ^b   Bahar, I ^a  

^a UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF PITTSBURGH   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

APOPTOSOME; CASPASE 3; INHIBITOR OF APOPTOSIS PROTEIN; PROTEIN BAX; PROTEIN BCL 2; CARRIER PROTEIN; ION CHANNEL; MITOCHONDRIAL PERMEABILITY TRANSITION PORE;

APOPTOSIS; ARTICLE; CELL MEMBRANE PERMEABILITY; CELL STIMULATION; CELL SURVIVAL; CHEMICAL REACTION KINETICS; ENZYME INHIBITION; EXAMINATION; MATHEMATICAL MODEL; MITOCHONDRION; MOLECULAR MECHANICS; PHASE TRANSITION; POROSITY; PREDICTION; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN STABILITY; PROTEIN SYNTHESIS; SIMULATION; ANIMAL; BIOLOGICAL MODEL; COMPUTER SIMULATION; HUMAN; METABOLISM; PHYSIOLOGY;

ANIMALS; APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CELL MEMBRANE PERMEABILITY; CELL SURVIVAL; COMPUTER SIMULATION; HUMANS; ION CHANNELS; MITOCHONDRIA; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MODELS, BIOLOGICAL; PROTO-ONCOGENE PROTEINS C-BCL-2;

EID: 33646124974     PISSN: 00063495     EISSN: None     Source Type: Journal    
DOI: 10.1529/biophysj.105.068122     Document Type: Article

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