Decreases in the precision of Purkinje cell pacemaking cause cerebellar dysfunction and ataxia

Nature Neuroscience

Volumn 9, Issue 3, 2006, Pages 389-397

  Walter, Joy T ^a   Alviña, Karina ^a   Womack, Mary D ^a   Chevez, Carolyn ^a   Khodakhah, Kamran ^a  

^a ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

1 ETHYL 2 BENZIMIDAZOLINONE; CALCIUM ACTIVATED POTASSIUM CHANNEL; CALCIUM CHANNEL P TYPE; CALCIUM CHANNEL Q TYPE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CELL ACTIVATION; CEREBELLAR ATAXIA; CONTROLLED STUDY; EVALUATION; MOTOR PERFORMANCE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PURKINJE CELL; SYNAPSE; SYNAPTIC TRANSMISSION;

ACTION POTENTIALS; ANIMALS; BENZIMIDAZOLES; BIOLOGICAL CLOCKS; CALCIUM CHANNEL AGONISTS; CALCIUM CHANNEL BLOCKERS; CALCIUM CHANNELS, P-TYPE; CEREBELLAR ATAXIA; CEREBELLAR CORTEX; DOWN-REGULATION; FEMALE; MALE; MICE; MICE, NEUROLOGIC MUTANTS; MICE, TRANSGENIC; POTASSIUM CHANNELS, CALCIUM-ACTIVATED; PURKINJE CELLS; SYNAPTIC MEMBRANES; SYNAPTIC TRANSMISSION;

EID: 33344454896     PISSN: 10976256     EISSN: None     Source Type: Journal    
DOI: 10.1038/nn1648     Document Type: Article

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