Enhanced neuronal excitability in the absence of neurodegeneration induces cerebellar ataxia

Journal of Clinical Investigation

Volumn 113, Issue 4, 2004, Pages 582-590

  Shakkottai, Vikram G ^a   Chou, Chin Hua ^a   Oddo, Salvatore ^a   Sailer, Claudia A ^b   Knaus, Hans Günther ^b   Gutman, George A ^c   Barish, Michael E ^d   LaFerla, Frank M ^a   Chandy, K George ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF INNSBRUCK   (Austria)

^c UNIVERSITY OF CALIFORNIA   (United States)

^d CITY OF HOPE NATIONAL MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

APAMIN; CALCIUM ACTIVATED POTASSIUM CHANNEL; GREEN FLUORESCENT PROTEIN; NEUROPROTECTIVE AGENT; RILUZOLE; HYBRID PROTEIN; POTASSIUM CHANNEL;

AFTERHYPERPOLARIZATION; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CEREBELLAR ATAXIA; CEREBELLUM NUCLEUS; DEEP CEREBELLUM NUCLEUS; DISEASE MODEL; GENE EXPRESSION; MOUSE; NERVE DEGENERATION; NERVE EXCITABILITY; NEUROPROTECTION; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; PURKINJE CELL; SPIKE WAVE; TRANSGENE; TRANSGENIC MOUSE; ANIMAL; CYTOLOGY; ELECTROPHYSIOLOGY; GENETICS; IN VITRO STUDY; METABOLISM; MOTOR ACTIVITY; NERVE CELL; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; PROMOTER REGION;

ANIMALS; APAMIN; CEREBELLAR ATAXIA; CEREBELLAR NUCLEI; ELECTROPHYSIOLOGY; MICE; MICE, TRANSGENIC; MOTOR ACTIVITY; NEURONS; POTASSIUM CHANNELS; PROMOTER REGIONS (GENETICS); PURKINJE CELLS; RECOMBINANT FUSION PROTEINS; TRANSGENES;

EID: 1642360155     PISSN: 00219738     EISSN: None     Source Type: Journal    
DOI: 10.1172/JCI200420216     Document Type: Article

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