Dual degradation signals control Gli protein stability and tumor formation

Genes and Development

Volumn 20, Issue 3, 2006, Pages 276-281

  Huntzicker, Erik G ^a   Estay, Ivette S ^a   Zhen, Hanson ^a   Lokteva, Ludmila A ^a   Jackson, Peter K ^a,b   Oro, Anthony E ^a  

^a STANFORD UNIVERSITY   (United States)

^b STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

TRCP; Basal cell carcinoma; Gli; Hair follicle; Hedgehog; Proteasome

Indexed keywords

PROTEASOME; TRANSCRIPTION FACTOR GLI1;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; BASAL CELL CARCINOMA; BIOACCUMULATION; CARCINOGENESIS; CONTROLLED STUDY; CORRELATION ANALYSIS; ENZYME ACTIVITY; HUMAN; HUMAN CELL; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; PROTEIN STABILITY; REGULATORY MECHANISM; SIGNAL TRANSDUCTION; TRANSGENIC ANIMAL;

AMINO ACID SEQUENCE; ANIMALS; CARCINOMA, BASAL CELL; CYTOPLASM; GENE EXPRESSION REGULATION; HUMANS; KERATINOCYTES; MICE; MICE, TRANSGENIC; MOLECULAR SEQUENCE DATA; MUTATION; NIH 3T3 CELLS; ONCOGENE PROTEINS; PROTEASOME ENDOPEPTIDASE COMPLEX; SIGNAL TRANSDUCTION; TRANS-ACTIVATORS; TRANSCRIPTION FACTORS; TRANSFECTION; XENOPUS;

ANIMALIA; ERINACEIDAE;

EID: 32044436273     PISSN: 08909369     EISSN: 15495477     Source Type: Journal    
DOI: 10.1101/gad.1380906     Document Type: Article

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