Impact of GSTT1, GSTM1, GSTP1 and NAT2 genotypes on KRAS2 and TP53 gene mutations in colorectal cancer

International Journal of Cancer

Volumn 110, Issue 2, 2004, Pages 183-187

  Ferraz, Jean Marc ^a   Zinzindohoué, Franck ^a   Lecomte, Thierry ^a   Cugnenc, Paul Henri ^a   Loriot, Marie Anne ^b   Beaune, Philippe ^b   Stücker, Isabelle ^b   Berger, Anne ^a   Laurent Puig, Pierre ^a,b,c  

^a HÔPITAL EUROPÉEN GEORGES POMPIDOU   (France)

^b INSERM   (France)

^c Lab de Toxicologie Moleculaire   (France)

Author keywords

Colorectal cancer; GSM1; GST1; GSTP1; KRAS2; NAT2; TP53

Indexed keywords

ADULT; AGED; ARTICLE; CANCER RISK; COLORECTAL CANCER; DIETARY INTAKE; DNA POLYMORPHISM; FEMALE; GENE; GENE FREQUENCY; GENE MUTATION; GENOTYPE; GSTM1 GENE; GSTP1 GENE; GSTT1 GENE; HUMAN; HUMAN TISSUE; HYPOTHESIS; KRAS2 GENE; MAJOR CLINICAL STUDY; MALE; NAT2 GENE; ONCOGENE K RAS; PRIORITY JOURNAL; TUMOR SUPPRESSOR GENE; WILD TYPE;

ADULT; AGED; AGED, 80 AND OVER; ARYLAMINE N-ACETYLTRANSFERASE; COLORECTAL NEOPLASMS; FEMALE; GENES, P53; GENES, RAS; GENOTYPE; GLUTATHIONE S-TRANSFERASE PI; GLUTATHIONE TRANSFERASE; HUMANS; ISOENZYMES; MALE; MIDDLE AGED; MUTATION; POLYMORPHISM, GENETIC;

EID: 2342483662     PISSN: 00207136     EISSN: None     Source Type: Journal    
DOI: 10.1002/ijc.20124     Document Type: Article

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